Effect of PGE2 and alpha-adrenergic agonists on AVP-dependent cAMP levels in rabbit and rat CCT

Am J Physiol. 1988 Jul;255(1 Pt 2):F43-8. doi: 10.1152/ajprenal.1988.255.1.F43.

Abstract

The effect of prostaglandins and alpha-adrenergic agonists on arginine vasopressin-induced adenosine 3',5'-cyclic monophosphate (cAMP) production was investigated in microdissected rat and rabbit cortical collecting tubules (CCT) incubated in vitro. In rabbit CCT, addition to all media of a prostaglandin synthesis inhibitor increased this production; exogenous prostaglandin E2 (PGE2) induced a reproducible dose-dependent inhibition of cAMP accumulation. Maximal inhibition (mean: 57.5%) was observed with 0.3 microM PGE2, and threshold inhibition was observed with concentrations ranging from 3 to 10 nM PGE2. Inhibition of cAMP levels in rabbit CCT was also obtained with 0.3 microM PGF2 alpha (mean inhibition: 44.3%) but not with alpha-adrenergic agonists studied under the same conditions. The opposite was observed in rat CCT studied in parallel: the alpha-agonists inhibited cAMP production by up to 80%, but PGE2 had no effect.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adrenergic alpha-Agonists
  • Animals
  • Arginine Vasopressin / pharmacology*
  • Clonidine / pharmacology
  • Cyclic AMP / metabolism*
  • Dinoprost
  • Dinoprostone
  • Kidney Tubules, Collecting
  • Male
  • Norepinephrine / pharmacology
  • Propranolol / pharmacology
  • Prostaglandins E / pharmacology
  • Prostaglandins F / pharmacology
  • Rabbits
  • Rats
  • Rats, Inbred Strains

Substances

  • Adrenergic alpha-Agonists
  • Prostaglandins E
  • Prostaglandins F
  • Arginine Vasopressin
  • Propranolol
  • Dinoprost
  • Cyclic AMP
  • Dinoprostone
  • Clonidine
  • Norepinephrine