T cell activation: distinct pathways involve phosphorylation of different cellular proteins

Eur J Immunol. 1988 Jul;18(7):1139-42. doi: 10.1002/eji.1830180727.

Abstract

The murine T cell clone D10.G4.1 can be induced to proliferate by monoclonal antibodies (mAb) to the T cell receptor (TcR) or to Thy-1 molecules. When cells were stimulated by anti-TcR mAb, a group of 4 proteins (19-25 kDa) was specifically phosphorylated. This effect was completely mimicked by the Ca2+ ionophore A23187, whereas only two of these proteins (19 kDa and 25 kDa) were phosphorylated after cell exposure to the phorbol ester 12-O-tetradecanoylphorbol 13-acetate. By contrast, anti-Thy-1 mAb had no effect on the phosphorylation of these proteins, but induced specifically the phosphorylation of a protein of 32 kDa. These results therefore demonstrate that distinct activating pathways in T cells involve the phosphorylation of different proteins, suggesting that the stimulation of protein kinases in T lymphocytes is an early event in cell activation.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Antibodies, Monoclonal / immunology
  • Antigens, Surface / physiology
  • Cell Line
  • Immunologic Techniques
  • Lymphocyte Activation*
  • Mice
  • Phosphoproteins / physiology*
  • Receptors, Antigen, T-Cell / physiology
  • T-Lymphocytes / physiology*
  • Thy-1 Antigens

Substances

  • Antibodies, Monoclonal
  • Antigens, Surface
  • Phosphoproteins
  • Receptors, Antigen, T-Cell
  • Thy-1 Antigens