Abstract
In 11 of 18 patients with human T-lymphotropic virus type-I (HTLV-I) associated myelopathy (HAM) gait, sensory, and/or sphincter disturbance improved with plasmapheresis (4 to 6 sessions in 2 weeks), and the effects were maintained for 2 to 4 weeks. Plasmapheresis lowered the titre of HTLV-I antibody in serum but not in cerebrospinal fluid, and change of HTLV-I antibody titres did not correlate with the effects of plasmapheresis. These results suggest that plasmapheresis is useful treatment, at least in producing a temporary improvement, in patients with HAM, and that some humoral factor(s), but not HTLV-I antibody, may be important in the pathogenesis of HAM.
Publication types
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Case Reports
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Research Support, Non-U.S. Gov't
MeSH terms
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Adult
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Aged
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Antibodies, Viral / analysis
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Antibodies, Viral / cerebrospinal fluid
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Anus Diseases / cerebrospinal fluid
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Anus Diseases / therapy
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Deltaretrovirus Infections / cerebrospinal fluid
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Deltaretrovirus Infections / immunology
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Deltaretrovirus Infections / therapy*
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Evaluation Studies as Topic
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Female
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Human T-lymphotropic virus 1 / immunology
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Humans
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Locomotion
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Male
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Middle Aged
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Movement Disorders / cerebrospinal fluid
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Movement Disorders / therapy
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Nervous System Diseases / cerebrospinal fluid
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Nervous System Diseases / therapy
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Plasmapheresis*
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Sensation
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Spinal Cord Diseases / cerebrospinal fluid
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Spinal Cord Diseases / immunology
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Spinal Cord Diseases / therapy*
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Time Factors