Response to: 'Acquiring new N- glycosylation sites in variable regions of immunoglobulin genes by somatic hypermutation is a common feature of autoimmune diseases' by Visser et al

Ann Rheum Dis. 2018 Oct;77(10):e70. doi: 10.1136/annrheumdis-2017-212583. Epub 2017 Nov 23.

Authors

Rochelle D Vergroesen¹, Linda M Slot¹, Lise Hafkenscheid¹, Marvyn T Koning², Hans U Scherer¹, René E M Toes¹

Affiliations

¹ Department of Rheumatology, Leiden University Medical Center, Leiden, The Netherlands.
² Department of Hematology, Leiden University Medical Center, Leiden, The Netherlands.

PMID: 29170200
DOI: 10.1136/annrheumdis-2017-212583

No abstract available

Keywords: autoimmune diseases; b cells; treatment.

Publication types

Letter
Comment

MeSH terms

Autoimmune Diseases*
B-Lymphocytes
Genes, Immunoglobulin*
Glycosylation
Humans
Immunoglobulin G
Receptors, Antigen, B-Cell

Substances

Immunoglobulin G
Receptors, Antigen, B-Cell