Cytokine gene polymorphism in denture stomatitis patients: A clinical study

Oral Dis. 2018 Sep;24(6):983-992. doi: 10.1111/odi.12844. Epub 2018 Jun 11.

Abstract

Objective: This clinical study investigated the association between cytokine gene polymorphism and Candida growth in denture stomatitis (DS) patients.

Subjects and methods: Saliva and blood samples of 160 complete denture wearers (80 healthy controls and 80 with DS) were collected for mycological and gene polymorphism testing, respectively. Salivary Candida growth and TNF-α, TGF-β, IL-6, and IL-10 genotypes were investigated. Data were analyzed using Student's t test, Mann-Whitney U test, chi-square analysis, and continuity (yates) correction tests (p < .05).

Results: Candida albicans colony counts in saliva were significantly higher in the DS group and in the TNF-α GG genotype (p < .05). TGF-β TC GG and TGF-β CC GG haplotypes were significantly higher in DS and control groups, respectively (p < .05). C. albicans colony counts were significantly higher in control group in the TGF-β TC GG haplotype (p < .05). Candida glabrata colony counts were significantly higher in the DS group than the control group in IL-6 GG genotype (p < .05). The difference between DS types in IL-6 genotypes was significant with lower expression level in DS type 3 than DS type 1 and also type 2 (p ≤ .01).

Conclusion: The significant differences in some genotypes of the TNF-α, TGF-β, and IL-6 in DS patients are promising in understanding the host defense in DS.

Keywords: Candida; cytokine; cytokine gene polymorphism; denture stomatitis.

Publication types

  • Clinical Study

MeSH terms

  • Aged
  • Candida albicans / growth & development*
  • Candida glabrata / growth & development*
  • Case-Control Studies
  • Colony Count, Microbial
  • Cytokines / genetics*
  • Dentures / adverse effects*
  • Female
  • Humans
  • Interleukin-10 / genetics
  • Interleukin-6 / genetics
  • Male
  • Middle Aged
  • Polymorphism, Genetic
  • Saliva / microbiology*
  • Stomatitis / etiology
  • Stomatitis / genetics*
  • Stomatitis / microbiology*
  • Transforming Growth Factor beta / genetics
  • Tumor Necrosis Factor-alpha / genetics

Substances

  • Cytokines
  • IL10 protein, human
  • IL6 protein, human
  • Interleukin-6
  • Transforming Growth Factor beta
  • Tumor Necrosis Factor-alpha
  • Interleukin-10