Host Cell Targeting by Enteropathogenic Bacteria T3SS Effectors

Trends Microbiol. 2018 Apr;26(4):266-283. doi: 10.1016/j.tim.2018.01.010. Epub 2018 Feb 21.

Abstract

Microbial pathogens possess a diversity of weapons that disrupt host homeostasis and immune defenses, thus resulting in the establishment of infection. The best-characterized system mediating bacterial protein delivery into target eukaryotic cells is the type III secretion system (T3SS) expressed by Gram-negative bacteria, including the human enteric pathogens Shigella, Salmonella, Yersinia, and enteropathogenic/enterohemorragic Escherichia coli (EPEC/EHEC). The emerging global view is that these T3SS-bearing pathogens share similarities in their ability to target key cellular pathways such as the cell cytoskeleton, trafficking, cell death/survival, and the NF-κB and MAPK signaling pathways. In particular, multiple host proteins are targeted in a given pathway, and different T3SS effectors from various pathogens share functional similarities.

Keywords: NF-κB and MAPK signaling pathways; cell cytoskeleton and trafficking; cell death and survival; host–pathogen cross-talk.

Publication types

  • Review

MeSH terms

  • Adaptive Immunity
  • Cytoskeleton
  • Enterohemorrhagic Escherichia coli / pathogenicity
  • Enteropathogenic Escherichia coli / pathogenicity
  • Homeostasis
  • Host-Pathogen Interactions / immunology*
  • Host-Pathogen Interactions / physiology*
  • Humans
  • Mitogen-Activated Protein Kinases
  • NF-kappa B
  • Protein Transport
  • Salmonella / pathogenicity
  • Shigella / pathogenicity
  • Type III Secretion Systems / physiology*
  • Yersinia / pathogenicity

Substances

  • NF-kappa B
  • Type III Secretion Systems
  • Mitogen-Activated Protein Kinases