Immunomodulatory treatment with systemic GM-CSF augments pulmonary immune responses and improves neurological outcome after experimental stroke

J Neuroimmunol. 2018 Aug 15:321:144-149. doi: 10.1016/j.jneuroim.2018.03.005. Epub 2018 Mar 9.

Abstract

Stroke-induced immunodepression is an independent risk factor for stroke-associated pneumonia (SAP). Granulocyte-macrophage colony stimulating factor (GM-CSF) has neuroprotective properties in experimental stroke and been demonstrated to reverse immunodepression in sepsis patients. However, whether GM-CSF restores immune function after stroke preventing SAP and improving outcome is unknown. Here, we demonstrated that GM-CSF treatment improved peripheral and pulmonary leukocyte numbers, peripheral cytokine responses, lowered lung bacterial burden in the early course and improved long-term functional outcome after experimental stroke. These data suggest that GM-CSF is promising for stroke treatment since it not only acts neuroprotective in the ischemic brain but may also protect against detrimental post-stroke infections.

Keywords: GM-CSF; Immunomodulation; MCAo; Neurological outcome; Pneumonia.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Granulocyte-Macrophage Colony-Stimulating Factor / administration & dosage*
  • Immunity, Cellular / drug effects
  • Immunity, Cellular / immunology
  • Immunologic Factors / administration & dosage
  • Lung / drug effects
  • Lung / immunology*
  • Male
  • Mice, Inbred C57BL
  • Nervous System Diseases / drug therapy*
  • Nervous System Diseases / etiology
  • Nervous System Diseases / immunology*
  • Stroke / complications
  • Stroke / drug therapy*
  • Stroke / immunology*
  • Treatment Outcome

Substances

  • Immunologic Factors
  • Granulocyte-Macrophage Colony-Stimulating Factor