3-Chloro-4,5-dihydroxybenzaldehyde inhibits adipogenesis in 3T3-L1 adipocytes by regulating expression of adipogenic transcription factors and AMPK activation

Chem Biol Interact. 2018 May 1:287:27-31. doi: 10.1016/j.cbi.2018.04.001. Epub 2018 Apr 6.

Abstract

Obesity is a serious health issue in many industrialized countries. It is a medical condition with excessive levels of fat accumulated in adipocytes. The objective of the present study was to determine the inhibitory effect of 3-chloro-4,5-dihydroxybenzaldehyde (CDB) on adipogenesis in 3T3-L1 adipocyte cells. CDB suppressed the differentiation and decreased lipid accumulation and triglycerides contents in 3T3-L1 adipocytes. Its suppression effect on fat accumulation was mediated via expression of adipogenesis factors (C/EBPα, SREBP-1c, PPARγ, and adiponectin) during adipocyte differentiation in white adipocyte cells. CDB's ability to suppress fat accumulation was increased in a concentration-dependent manner. It inhibited fatty acid synthesis related proteins including FAS, FABP4, leptin, and perilipin. It also increased expression of phosphorylated AMPK in adipocytes cells. These observations suggest that CDB has potential anti-obesity effect with ability to improve metabolic diseases.

Keywords: 3-Chloro-4,5-dihydroxybenzaldehyde; 3T3-L1; Adipocytes; Adipogenesis; Anti-obesity.

MeSH terms

  • 3T3-L1 Cells
  • AMP-Activated Protein Kinases / metabolism*
  • Adipocytes / cytology
  • Adipocytes / drug effects
  • Adipocytes / metabolism
  • Adipogenesis / drug effects*
  • Adiponectin / metabolism
  • Animals
  • Anti-Obesity Agents / chemistry
  • Anti-Obesity Agents / pharmacology*
  • Benzaldehydes / chemistry
  • Benzaldehydes / pharmacology*
  • CCAAT-Enhancer-Binding Protein-alpha / metabolism
  • Cell Survival / drug effects
  • Enzyme Activation / drug effects
  • Fatty Acid-Binding Proteins / metabolism
  • Gene Expression Regulation / drug effects*
  • Leptin / metabolism
  • Mice
  • PPAR gamma / metabolism
  • Sterol Regulatory Element Binding Protein 1 / metabolism
  • Transcription Factors / genetics*
  • Triglycerides / metabolism
  • fas Receptor / metabolism

Substances

  • 3-chloro-4,5-dihydroxybenzaldehyde
  • Adiponectin
  • Anti-Obesity Agents
  • Benzaldehydes
  • CCAAT-Enhancer-Binding Protein-alpha
  • Fabp4 protein, mouse
  • Fas protein, mouse
  • Fatty Acid-Binding Proteins
  • Leptin
  • PPAR gamma
  • Sterol Regulatory Element Binding Protein 1
  • Transcription Factors
  • Triglycerides
  • fas Receptor
  • AMP-Activated Protein Kinases