A group of tissue-specific microRNAs contribute to the silencing of CUX1 in different cell lineages during development

J Cell Biochem. 2018 Jul;119(7):6238-6248. doi: 10.1002/jcb.26852. Epub 2018 Apr 16.

Abstract

Cut-like homeobox 1 (CUX1) is a highly conserved homeoprotein that functions as a transcriptional repressor of genes specifying terminal differentiation. We previously showed that liver-specific microRNA-122 (miR-122) regulates the timing of liver development by silencing CUX1 post-transcriptionally. Since the CUX1 protein is expressed in a subset of embryonic tissues, we hypothesized that it is regulated by specific microRNAs (miRNAs) in each cell type during development. Using a large-scale screening method, we identified ten tissue-specific miRNAs from different cell lineages that directly targeted CUX1. An analysis of the interaction between heart-specific microRNA-208a (miR-208a) and CUX1 in the hearts of developing mouse embryos and in P19CL6 cells undergoing cardiac differentiation indicated that CUX1 is regulated by miR-208a during heart development and cardiomyocyte differentiation. Functional analysis of miR-208a in P19CL6 cells using lentiviral-mediated over-expression showed that it regulates the transition between cellular proliferation and differentiation. These results suggest that these tissue-specific miRNAs might play a common role in timing the progression of terminal differentiation of different cell lineages, possibly by silencing the differentiation repressor CUX1.

Keywords: CUX1; differentiation; lineage; miRNA; tissue-specific.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Cell Differentiation*
  • Cell Lineage / genetics*
  • Cell Proliferation
  • Cells, Cultured
  • Gene Expression Regulation, Developmental*
  • HeLa Cells
  • Heart / growth & development
  • Homeodomain Proteins / antagonists & inhibitors*
  • Humans
  • Male
  • Mice
  • Mice, Inbred C57BL
  • MicroRNAs / genetics*
  • Myocytes, Cardiac / cytology*
  • Myocytes, Cardiac / metabolism
  • Nuclear Proteins / antagonists & inhibitors*
  • Organ Specificity
  • Repressor Proteins / antagonists & inhibitors*
  • Transcription Factors

Substances

  • CUX1 protein, human
  • Cux1 protein, mouse
  • Homeodomain Proteins
  • MicroRNAs
  • Nuclear Proteins
  • Repressor Proteins
  • Transcription Factors