The small RNA complement of adult Schistosoma haematobium

PLoS Negl Trop Dis. 2018 May 29;12(5):e0006535. doi: 10.1371/journal.pntd.0006535. eCollection 2018 May.

Abstract

Background: Blood flukes of the genus Schistosoma cause schistosomiasis-a neglected tropical disease (NTD) that affects more than 200 million people worldwide. Studies of schistosome genomes have improved our understanding of the molecular biology of flatworms, but most of them have focused largely on protein-coding genes. Small non-coding RNAs (sncRNAs) have been explored in selected schistosome species and are suggested to play essential roles in the post-transcriptional regulation of genes, and in modulating flatworm-host interactions. However, genome-wide small RNA data are currently lacking for key schistosomes including Schistosoma haematobium-the causative agent of urogenital schistosomiasis of humans.

Methodology: MicroRNAs (miRNAs) and other sncRNAs of male and female adults of S. haematobium and small RNA transcription levels were explored by deep sequencing, genome mapping and detailed bioinformatic analyses.

Principal findings: In total, 89 transcribed miRNAs were identified in S. haematobium-a similar complement to those reported for the congeners S. mansoni and S. japonicum. Of these miRNAs, 34 were novel, with no homologs in other schistosomes. Most miRNAs (n = 64) exhibited sex-biased transcription, suggestive of roles in sexual differentiation, pairing of adult worms and reproductive processes. Of the sncRNAs that were not miRNAs, some related to the spliceosome (n = 21), biogenesis of other RNAs (n = 3) or ribozyme functions (n = 16), whereas most others (n = 3798) were novel ('orphans') with unknown functions.

Conclusions: This study provides the first genome-wide sncRNA resource for S. haematobium, extending earlier studies of schistosomes. The present work should facilitate the future curation and experimental validation of sncRNA functions in schistosomes to enhance our understanding of post-transcriptional gene regulation and of the roles that sncRNAs play in schistosome reproduction, development and parasite-host cross-talk.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Computational Biology
  • Cricetinae
  • Female
  • Gene Expression Regulation
  • Humans
  • Male
  • RNA, Complementary / genetics*
  • RNA, Complementary / metabolism
  • RNA, Small Untranslated / genetics*
  • RNA, Small Untranslated / metabolism
  • Schistosoma haematobium / genetics*
  • Schistosoma haematobium / metabolism
  • Schistosomiasis haematobia / parasitology*
  • Sequence Analysis, RNA

Substances

  • RNA, Complementary
  • RNA, Small Untranslated

Grants and funding

The present study was supported by funding from the National Health and Medical Research Council of Australia (NHMRC) and the Australian Research Council (ARC) to RBG and NDY. Additional support from the Alexander von Humboldt Foundation (Germany), Melbourne Water Corporation (Australia), Yourgene Bioscience (Taiwan) to RBG. Career Development Fellowships to NDY and ARJ, and Early Career Research Fellowship to PKK were from NHMRC. The funders had no role in study design, data collection and analysis, decision to publish, or preparation of the manuscript.