Growth-promoting effect of gastrin on human gastric carcinoma cell line TMK-1

Jpn J Cancer Res. 1985 Nov;76(11):1064-71.

Abstract

A human gastric carcinoma cell line TMK-1 was established in vitro by the soft agar method from SC-6-JCK, a poorly differentiated adenocarcinoma xenotransplanted in nude mice. TMK-1 cells had a doubling time of approximately 35 hr and showed carcinoembryonic antigen (CEA), alpha 1-antitrypsin and secretory component immunoreactivity. Ultrastructurally, the tumor cells were characterized by numerous mitochondria, tubulovesicles and intracytoplasmic canaliculi filled with abundant microvilli. The growth of TMK-1 cells was promoted by 10nM human gastrin (G-17), 2 microM tetragastrin or 2 microM pentagastrin, among which human gastrin showed the most effective growth promotion. Moreover, incorporation of [3H]thymidine into TMK-1 cells was stimulated by gastrin in a dose-dependent manner. The content of cyclic adenosine 3',5'-monophosphate (cAMP) in TMK-1 cells was increased by gastrin treatment but decreased to the control level within 10 min. cAMP-dependent protein kinase was also activated by gastrin administration.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Carcinoma / enzymology
  • Carcinoma / pathology*
  • Cell Cycle / drug effects
  • Cell Line
  • Cyclic AMP / metabolism
  • DNA, Neoplasm / biosynthesis
  • Dose-Response Relationship, Drug
  • Gastrins / pharmacology*
  • Humans
  • Microscopy, Electron
  • Protein Kinases / metabolism
  • Stomach Neoplasms / enzymology
  • Stomach Neoplasms / pathology*
  • Time Factors

Substances

  • DNA, Neoplasm
  • Gastrins
  • Cyclic AMP
  • Protein Kinases