Attenuated adenosine R-site effect in adipocytes in obesity

Metabolism. 1986 Feb;35(2):143-6. doi: 10.1016/0026-0495(86)90115-0.

Abstract

Adenosine is a local hormone or a retaliatory metabolite that executes its effect via a plasma membrane receptor, the R-site. In human adipocytes it inhibits cyclic AMP accumulation and lipolysis. N6-(phenylisopropyl)adenosine is a nonmetabolizable derivative that is an R-site agonist not sharing other effects of the parent nucleoside. In subcutaneous abdominal fat cells from obese subjects (130% to 207% of ideal body weight, N = 8), the antilipolytic effect of N6-(phenylisopropyl) adenosine was markedly attenuated as compared to that in fat cells from normal weight subjects (83% to 121% of ideal body weight, N = 8). There was a negative correlation between the effectiveness of the nucleoside analog and the relative body weight of the donor. The effect of N6-(phenylisopropyl)adenosine on cyclic AMP accumulation was similarly attenuated. These findings may explain some of the metabolic alterations observed in obesity.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adenosine / metabolism
  • Adipose Tissue / metabolism*
  • Body Weight
  • Cyclic AMP / metabolism
  • Humans
  • Isoproterenol / pharmacology
  • Lipolysis
  • Obesity / physiopathology*
  • Phenylisopropyladenosine / metabolism
  • Receptors, Cell Surface / physiology*
  • Receptors, Purinergic

Substances

  • Receptors, Cell Surface
  • Receptors, Purinergic
  • Phenylisopropyladenosine
  • Cyclic AMP
  • Adenosine
  • Isoproterenol