Transcription of the human beta-globin gene is stimulated by an SV40 enhancer to which it is physically linked but topologically uncoupled

Cell. 1986 May 23;45(4):575-80. doi: 10.1016/0092-8674(86)90289-8.

Abstract

One postulated mechanism for how the SV40 enhancer stimulates transcription of linked genes involves the enhancer as a binding site for a sequence-specific "gyrase" activity. We sought to test this hypothesis directly by constructing a novel heteroduplex circle, termed a tailed-circle, in which one of the strands contains an extra palindromic sequence base-paired into a hairpin structure. The human beta-globin gene is placed in the circle and the SV40 enhancer on the hairpin tail, where a bound topoisomerase cannot supercoil the circle. Upon transfection of this DNA into HeLa cells the SV40 enhancer on the hairpin arm is still able to stimulate transcription of the beta-globin gene. Southern blot analysis of the DNA after transfection does not demonstrate any repair or replication of the tailed-circle in vivo. These results argue against the sequence-specific gyrase model for SV40 enhancer action.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • DNA Topoisomerases, Type II / metabolism*
  • DNA, Recombinant / metabolism
  • DNA, Superhelical / metabolism
  • Enhancer Elements, Genetic*
  • Genes, Regulator*
  • Genes, Synthetic
  • Globins / biosynthesis*
  • Globins / genetics
  • HeLa Cells
  • Humans
  • Nucleic Acid Conformation
  • Plasmids
  • Recombinant Proteins / biosynthesis
  • Recombinant Proteins / genetics
  • Simian virus 40 / genetics*
  • Transcription, Genetic*

Substances

  • DNA, Recombinant
  • DNA, Superhelical
  • Recombinant Proteins
  • Globins
  • DNA Topoisomerases, Type II