Coxsackievirus B3 infection alters plasma membrane of neonatal skin fibroblasts

J Virol. 1986 Oct;60(1):294-6. doi: 10.1128/JVI.60.1.294-296.1986.

Abstract

Replication of coxsackievirus B3 occurred for days in cultures of murine neonatal skin fibroblasts in the absence of cytopathology and resulted in alteration of the plasma membrane. Dual immunofluorescence studies showed that the lectin Ulex europaeus agglutinin I bound only to cells producing viral capsid antigens. Cultures of coxsackievirus B3-inoculated murine neonatal skin fibroblasts showed maximum binding of this lectin at 72 h postinoculation. These data show that in a nonlytic infection a picornavirus can alter the surface of an infected cell.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Animals
  • Animals, Newborn
  • Cell Membrane / ultrastructure*
  • Cells, Cultured
  • Cytopathogenic Effect, Viral
  • Enterovirus B, Human / physiology*
  • Fibroblasts / metabolism
  • Fibroblasts / microbiology
  • Fibroblasts / ultrastructure*
  • Lectins / metabolism
  • Mice
  • Plant Lectins*
  • Virus Replication

Substances

  • Lectins
  • Plant Lectins
  • Ulex europaeus lectins