Subcutaneous infection with dematiaceous fungi in Card9 knockout mice reveals association of impair neutrophils and Th cell response

J Dermatol Sci. 2018 Nov;92(2):215-218. doi: 10.1016/j.jdermsci.2018.08.005. Epub 2018 Aug 24.

Authors

Weiwei Wu¹, Ruijun Zhang¹, Xiaowen Wang¹, Yinggai Song¹, Ruoyu Li²

Affiliations

¹ Department of Dermatology, Peking UniversityFirst Hospital, Research Center for Medical Mycology, Peking University, Beijing Key Laboratory of Molecular Diagnosis on Dermatoses, Beijing, China.
² Department of Dermatology, Peking UniversityFirst Hospital, Research Center for Medical Mycology, Peking University, Beijing Key Laboratory of Molecular Diagnosis on Dermatoses, Beijing, China. Electronic address: [email protected].

PMID: 30190097
DOI: 10.1016/j.jdermsci.2018.08.005

No abstract available

Publication types

Letter

MeSH terms

Animals
CARD Signaling Adaptor Proteins / deficiency*
CARD Signaling Adaptor Proteins / genetics
Candidiasis, Chronic Mucocutaneous / genetics
Candidiasis, Chronic Mucocutaneous / immunology*
Candidiasis, Chronic Mucocutaneous / metabolism
Dermatomycoses / genetics
Dermatomycoses / immunology*
Dermatomycoses / metabolism
Dermatomycoses / microbiology
Disease Models, Animal
Host-Pathogen Interactions
Immunocompromised Host
Mice, Inbred C57BL
Mice, Knockout
Neutrophils / immunology*
Neutrophils / metabolism
Neutrophils / microbiology
Phaeohyphomycosis
Skin / immunology*
Skin / metabolism
Skin / microbiology
T-Lymphocytes, Helper-Inducer / immunology*
T-Lymphocytes, Helper-Inducer / metabolism
T-Lymphocytes, Helper-Inducer / microbiology
Time Factors

Substances

CARD Signaling Adaptor Proteins
Card9 protein, mouse

Supplementary concepts

Candidiasis familial chronic mucocutaneous, autosomal recessive