TANGO2: expanding the clinical phenotype and spectrum of pathogenic variants

Genet Med. 2019 Mar;21(3):601-607. doi: 10.1038/s41436-018-0137-y. Epub 2018 Sep 24.

Abstract

Purpose: TANGO2-related disorders were first described in 2016 and prior to this publication, only 15 individuals with TANGO2-related disorder were described in the literature. Primary features include metabolic crisis with rhabdomyolysis, encephalopathy, intellectual disability, seizures, and cardiac arrhythmias. We assess whether genotype and phenotype of TANGO2-related disorder has expanded since the initial discovery and determine the efficacy of exome sequencing (ES) as a diagnostic tool for detecting variants.

Methods: We present a series of 14 individuals from 11 unrelated families with complex medical and developmental histories, in whom ES or microarray identified compound heterozygous or homozygous variants in TANGO2.

Results: The initial presentation of patients with TANGO2-related disorders can be variable, including primarily neurological presentations. We expand the phenotype and genotype for TANGO2, highlighting the variability of the disorder.

Conclusion: TANGO2-related disorders can have a more diverse clinical presentation than previously anticipated. We illustrate the utility of routine ES data reanalysis whereby discovery of novel disease genes can lead to a diagnosis in previously unsolved cases and the need for additional copy-number variation analysis when ES is performed.

Keywords: developmental delay DNA copy-number variation; epilepsy; exome sequencing; intragenic deletion.

Publication types

  • Research Support, N.I.H., Extramural
  • Research Support, U.S. Gov't, Non-P.H.S.

MeSH terms

  • Adolescent
  • Aryl Hydrocarbon Receptor Nuclear Translocator / genetics*
  • Aryl Hydrocarbon Receptor Nuclear Translocator / physiology
  • Brain Diseases / genetics
  • Child
  • Child, Preschool
  • DNA Copy Number Variations / genetics
  • Developmental Disabilities / genetics
  • Exome
  • Exome Sequencing / methods
  • Family
  • Female
  • Genotype
  • Humans
  • Intellectual Disability / genetics
  • Male
  • Pedigree
  • Phenotype
  • Seizures / genetics

Substances

  • Aryl Hydrocarbon Receptor Nuclear Translocator