High fat diet reduces the expression of miRNA-29b in heart and increases susceptibility of myocardium to ischemia/reperfusion injury

J Cell Physiol. 2019 Jun;234(6):9399-9407. doi: 10.1002/jcp.27624. Epub 2018 Oct 26.

Abstract

Several studies have shown the role of microRNAs (miRNAs) in myocardial dysfunction in response to ischemia/reperfusion (I/R). In this study, we investigated the impact of high fat (HF) diet in the myocardial susceptibility to I/R injury, as well as in the expression of miRNA-29b. Isolated heart experiments using the ex vivo Langendorff perfusion model were used to induce cardiac I/R injury. HF diet-induced cardiac hypertrophy and impaired cardiac functional recovery after I/R. miRNA-29b, which targets Col1, was reduced in the heart of HF diet-fed mice, whereas the cardiac expression of Col1 was increased. In addition, hypoxia-reoxygenation (H/R) reduced the expression of miRNA-29b in cardiomyoblasts cultures. However, the overexpression of miRNA-29b in cardiomyoblasts reduced p53 mRNA levels and H/R injury, suggesting that downregulation of miRNA-29b may be involved in I/R injury. Together, our findings suggest that the reduced expression of miRNA-29b may be involved in the deteriorated cardiac functional recovery following I/R in obese mice.

Keywords: heart; hypoxia; ischemia/reperfusion injury; miRNA-29b; obesity.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Body Weight
  • Cell Line
  • Collagen / genetics
  • Collagen / metabolism
  • Diet, High-Fat*
  • Dyslipidemias / complications
  • Dyslipidemias / pathology
  • Glucose Intolerance / complications
  • Glucose Intolerance / pathology
  • Heart Function Tests
  • Hypoxia / complications
  • Hypoxia / genetics
  • Hypoxia / pathology
  • L-Lactate Dehydrogenase / metabolism
  • Male
  • Mice, Inbred C57BL
  • MicroRNAs / genetics*
  • MicroRNAs / metabolism
  • Myocardial Reperfusion Injury / complications
  • Myocardial Reperfusion Injury / genetics*
  • Myocardial Reperfusion Injury / pathology
  • Myocardial Reperfusion Injury / physiopathology
  • Myocardium / metabolism*
  • Myocardium / pathology*

Substances

  • MIRN29 microRNA, mouse
  • MicroRNAs
  • Collagen
  • L-Lactate Dehydrogenase