Draxin alters laminin organization during basement membrane remodeling to control cranial neural crest EMT

Dev Biol. 2019 Feb 15;446(2):151-158. doi: 10.1016/j.ydbio.2018.12.021. Epub 2018 Dec 21.

Abstract

Premigratory neural crest cells arise within the dorsal neural tube and subsequently undergo an epithelial-to-mesenchymal transition (EMT) to leave the neuroepithelium and initiate migration. Draxin is a Wnt modulator that has been shown to control the timing of cranial neural crest EMT. Here we show that this process is accompanied by three stages of remodeling of the basement membrane protein laminin, from regression to expansion and channel formation. Loss of Draxin results in blocking laminin remodeling at the regression stage, whereas ectopic maintenance of Draxin blocks remodeling at the expansion stage. The latter effect is rescued by addition of Snail2, previously shown to be downstream of Draxin. Our results demonstrate an essential function for the Wnt modulator Draxin in regulating basement membrane remodeling during cranial neural crest EMT.

Keywords: Draxin; Epithelial-to-Mesenchymal Transition; Laminin; Neural crest; Snail2.

Publication types

  • Research Support, N.I.H., Extramural

MeSH terms

  • Animals
  • Avian Proteins / genetics*
  • Avian Proteins / metabolism
  • Basement Membrane / embryology
  • Basement Membrane / metabolism*
  • Cell Movement / genetics
  • Chick Embryo
  • Epithelial-Mesenchymal Transition / genetics*
  • Gene Expression Regulation, Developmental*
  • Gene Knockdown Techniques
  • Laminin / genetics*
  • Laminin / metabolism
  • Neural Crest / embryology
  • Neural Crest / metabolism*
  • Skull / embryology
  • Snail Family Transcription Factors / genetics
  • Snail Family Transcription Factors / metabolism

Substances

  • Avian Proteins
  • Laminin
  • Snail Family Transcription Factors