Abstract
Background:
We performed profiling of the immune microenvironment of castration-resistant (CRPC) and castration-sensitive (CSPC) prostate cancer (PC) in order to identify novel targets for immunotherapy.
Methods:
PD-L1 and CD3/CD8 immunohistochemistry, PD-L1/2 fluorescent in situ hybridization, tumor mutation burden, microsatellite instability, and RNA-seq of 395 immune-related genes were performed in 19 CRPC and CSPC. Targeted genomic sequencing and fusion analysis were performed in 17 of these specimens.
Results:
CD276, PVR, and NECTIN2 were highly expressed in PC. Comparison of CRPC versus CSPC and primary versus metastatic tissue revealed the differential expression of immunostimulatory, immunosuppressive, and epithelial-to-mesenchymal transition (EMT)-related genes. Unsupervised clustering of differentially expressed genes yielded two final clusters best segregated by CRPC and CSPC status.
Conclusion:
CD276 and the alternative checkpoint inhibition PVR/NECTIN2/CD226/TIGIT pathway emerged as relevant to PC checkpoint inhibition target development.
Keywords:
CD226; CD276; NECTIN2; PVR; TIGIT; aggressive prostate cancer; castration-resistant prostate cancer; castration-sensitive prostate cancer; immunotherapy.
© 2019 Wiley Periodicals, Inc.
Publication types
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Research Support, Non-U.S. Gov't
MeSH terms
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Aged
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Antigens, CD / biosynthesis
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Antigens, CD / genetics
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Antigens, CD / immunology
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B7-H1 Antigen / biosynthesis
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B7-H1 Antigen / genetics
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B7-H1 Antigen / immunology
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Humans
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Immunohistochemistry
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Immunotherapy / methods
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Male
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Microsatellite Instability
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Middle Aged
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Nectins / biosynthesis
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Nectins / genetics
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Nectins / immunology
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Programmed Cell Death 1 Ligand 2 Protein / biosynthesis
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Programmed Cell Death 1 Ligand 2 Protein / genetics
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Programmed Cell Death 1 Ligand 2 Protein / immunology
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Prostatic Neoplasms / genetics
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Prostatic Neoplasms / immunology*
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Prostatic Neoplasms / therapy*
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Prostatic Neoplasms, Castration-Resistant / genetics
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Prostatic Neoplasms, Castration-Resistant / immunology*
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Prostatic Neoplasms, Castration-Resistant / therapy*
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RNA, Neoplasm / biosynthesis
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RNA, Neoplasm / genetics
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RNA, Neoplasm / immunology
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TOR Serine-Threonine Kinases / biosynthesis
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TOR Serine-Threonine Kinases / genetics
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TOR Serine-Threonine Kinases / immunology
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Tumor Microenvironment / immunology
Substances
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Antigens, CD
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B7-H1 Antigen
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CD274 protein, human
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NECTIN2 protein, human
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Nectins
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PDCD1LG2 protein, human
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Programmed Cell Death 1 Ligand 2 Protein
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RNA, Neoplasm
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MTOR protein, human
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TOR Serine-Threonine Kinases