Regulation of interleukin 2 receptors on T cells from multiple sclerosis patients

Proc Natl Acad Sci U S A. 1986 Apr;83(8):2637-41. doi: 10.1073/pnas.83.8.2637.

Abstract

Receptors for interleukin 2 (IL-2) are absent on resting T lymphocytes and are induced by antigenic and mitogenic stimulation. After a limited time (8-12 days), these receptors on normal T cells are down-regulated despite the presence of receptor-saturating concentrations of IL-2. We report here that both antigen- and mitogen-induced T-cell lines and clones obtained from peripheral blood and cerebrospinal fluid of multiple sclerosis patients show prolonged expression of IL-2 receptors. This expression is coincident with a prolonged responsiveness to the proliferative effects of IL-2. In addition, Leu 3+, IL-2 receptor-positive T-cell clone from the cerebrospinal fluid of a multiple sclerosis patient has been established and maintained for more than 1 year without IL-2. This clone has some morphologic and histochemical properties of T cells transformed or infected by human T-lymphotropic virus type I.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Antibodies, Monoclonal
  • Antigens, Differentiation, T-Lymphocyte
  • Antigens, Surface / analysis
  • Cerebrospinal Fluid / cytology
  • Humans
  • Interleukin-2 / pharmacology*
  • Lymphocyte Activation
  • Multiple Sclerosis / cerebrospinal fluid
  • Multiple Sclerosis / immunology*
  • Receptors, Immunologic / physiology*
  • Receptors, Interleukin-2
  • Recombinant Proteins / pharmacology*
  • T-Lymphocytes / immunology*

Substances

  • Antibodies, Monoclonal
  • Antigens, Differentiation, T-Lymphocyte
  • Antigens, Surface
  • Interleukin-2
  • Receptors, Immunologic
  • Receptors, Interleukin-2
  • Recombinant Proteins