MRI Imaging of the Hemodynamic Vasculature of Neuroblastoma Predicts Response to Antiangiogenic Treatment

Cancer Res. 2019 Jun 1;79(11):2978-2991. doi: 10.1158/0008-5472.CAN-18-3412. Epub 2019 Mar 15.

Abstract

Childhood neuroblastoma is a hypervascular tumor of neural origin, for which antiangiogenic drugs are currently being evaluated; however, predictive biomarkers of treatment response, crucial for successful delivery of precision therapeutics, are lacking. We describe an MRI-pathologic cross-correlative approach using intrinsic susceptibility (IS) and susceptibility contrast (SC) MRI to noninvasively map the vascular phenotype in neuroblastoma Th-MYCN transgenic mice treated with the vascular endothelial growth factor receptor inhibitor cediranib. We showed that the transverse MRI relaxation rate R 2* (second-1) and fractional blood volume (fBV, %) were sensitive imaging biomarkers of hemorrhage and vascular density, respectively, and were also predictive biomarkers of response to cediranib. Comparison with MRI and pathology from patients with MYCN-amplified neuroblastoma confirmed the high degree to which the Th-MYCN model vascular phenotype recapitulated that of the clinical phenotype, thereby supporting further evaluation of IS- and SC-MRI in the clinic. This study reinforces the potential role of functional MRI in delivering precision medicine to children with neuroblastoma. SIGNIFICANCE: This study shows that functional MRI predicts response to vascular-targeted therapy in a genetically engineered murine model of neuroblastoma.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Angiogenesis Inhibitors / pharmacology*
  • Animals
  • Child
  • Child, Preschool
  • Contrast Media
  • Female
  • Humans
  • Infant
  • Magnetic Resonance Imaging / methods*
  • Male
  • Mice, Transgenic
  • N-Myc Proto-Oncogene Protein / genetics
  • Neoplasms, Experimental
  • Neuroblastoma / blood supply
  • Neuroblastoma / diagnostic imaging*
  • Neuroblastoma / drug therapy*
  • Prospective Studies
  • Protein Kinase Inhibitors / pharmacology
  • Quinazolines / pharmacology*
  • Treatment Outcome

Substances

  • Angiogenesis Inhibitors
  • Contrast Media
  • MYCN protein, human
  • N-Myc Proto-Oncogene Protein
  • Protein Kinase Inhibitors
  • Quinazolines
  • cediranib