Predictive Biomarkers for Checkpoint Inhibitor-Based Immunotherapy: The Galectin-3 Signature in NSCLCs

Int J Mol Sci. 2019 Mar 31;20(7):1607. doi: 10.3390/ijms20071607.

Abstract

Checkpoint inhibitor-based immunotherapy is opening a promising scenario in oncology, with objective responses registered in multiple cancer types. However, reliable predictive markers of tumor responsiveness are still lacking. These markers need to be urgently identified for a better selection of patients that can be candidates for immunotherapy. In this pilot study, a cohort of 34 consecutive patients bearing programmed death-ligand 1 (PD-L1)-positive non-small cell lung carcinoma (NSCLC), treated with pembrolizumab, was considered. The retrospective immuno-phenotypic analysis performed on the original tumor biopsies allowed for the identification of a specific "galectin signature", which strongly correlated with tumor responsiveness to anti PD-1 immunotherapy. We observed that the large majority of patients (about 90%) with high galectin-3 tumor expression (score 3+) showed an early and dramatic progression of the disease after three cycles of treatments. In contrast, all patients with negative or low/intermediate expression of galectin-3 in tumor cells showed an early and durable objective response to pembrolizumab, indicating galectin-3 as an interesting predictive marker of tumor responsiveness. The galectin-3 signature, at least in NSCLCs, promises a better selection of patient candidates for immunotherapy, reducing unnecessary treatment exposures and social costs. A large multicenter study is ongoing to validate this finding.

Keywords: checkpoint inhibitors; galectin-3; non-small cell lung carcinoma; pembrolizumab; predictive marker.

Publication types

  • Clinical Trial

MeSH terms

  • Aged
  • Antibodies, Monoclonal, Humanized / therapeutic use
  • Antineoplastic Agents, Immunological / therapeutic use
  • Biomarkers, Tumor / metabolism*
  • Blood Proteins
  • Carcinoma, Non-Small-Cell Lung / drug therapy*
  • Carcinoma, Non-Small-Cell Lung / metabolism*
  • Galectin 3 / metabolism*
  • Galectins
  • Humans
  • Immunohistochemistry
  • Immunotherapy
  • Lung Neoplasms / drug therapy*
  • Lung Neoplasms / metabolism*
  • Middle Aged
  • Programmed Cell Death 1 Receptor / metabolism
  • Retrospective Studies

Substances

  • Antibodies, Monoclonal, Humanized
  • Antineoplastic Agents, Immunological
  • Biomarkers, Tumor
  • Blood Proteins
  • Galectin 3
  • Galectins
  • LGALS3 protein, human
  • Programmed Cell Death 1 Receptor
  • pembrolizumab