Cutting Edge: BCAP Promotes Lupus-like Disease and TLR-Mediated Type I IFN Induction in Plasmacytoid Dendritic Cells

J Immunol. 2019 May 1;202(9):2529-2534. doi: 10.4049/jimmunol.1801267. Epub 2019 Apr 1.

Abstract

Systemic lupus erythematosus severity correlates with elevated serum levels of type I IFNs, cytokines produced in large quantities by plasmacytoid dendritic cells (pDC) in response to engagement of TLR7 and TLR9 with endocytosed nucleic acids. B cell adaptor for PI3K (BCAP) promoted many aspects of TLR7-driven lupus-like disease, including Isg15 and Ifit1 expression in blood and an immature pDC phenotype associated with higher IFN production. BCAP-/- mice produced significantly less serum IFN-α than wild-type mice after injection of TLR9 agonist, and BCAP promoted TLR7 and TLR9-induced IFN-α production specifically in pDC. TLR-induced IFN-α production in pDC requires DOCK2-mediated activation of Rac1 leading to activation of IKKα, a mechanism we show was dependent on BCAP. BCAP-/- pDC had decreased actin polymerization and Rac1 activation and reduced IKKα phosphorylation upon TLR9 stimulation. We show a novel role for BCAP in promoting TLR-induced IFN-α production in pDC and in systemic lupus erythematosus pathogenesis.

Publication types

  • Research Support, N.I.H., Extramural

MeSH terms

  • Adaptor Proteins, Signal Transducing / genetics
  • Adaptor Proteins, Signal Transducing / immunology*
  • Animals
  • Cytokines / genetics
  • Cytokines / immunology
  • Dendritic Cells / immunology*
  • Dendritic Cells / pathology
  • Female
  • GTPase-Activating Proteins / genetics
  • GTPase-Activating Proteins / immunology
  • Guanine Nucleotide Exchange Factors / genetics
  • Guanine Nucleotide Exchange Factors / immunology
  • Interferon-alpha / genetics
  • Interferon-alpha / immunology*
  • Lupus Erythematosus, Systemic / genetics
  • Lupus Erythematosus, Systemic / immunology*
  • Lupus Erythematosus, Systemic / pathology
  • Male
  • Membrane Glycoproteins / genetics
  • Membrane Glycoproteins / immunology*
  • Mice
  • Mice, Knockout
  • Neuropeptides / genetics
  • Neuropeptides / immunology
  • Plasma Cells / immunology*
  • Plasma Cells / pathology
  • RNA-Binding Proteins / genetics
  • RNA-Binding Proteins / immunology
  • Toll-Like Receptor 7 / genetics
  • Toll-Like Receptor 7 / immunology*
  • Toll-Like Receptor 9 / genetics
  • Toll-Like Receptor 9 / immunology*
  • Ubiquitins / genetics
  • Ubiquitins / immunology
  • rac1 GTP-Binding Protein / genetics
  • rac1 GTP-Binding Protein / immunology

Substances

  • Adaptor Proteins, Signal Transducing
  • Cytokines
  • DOCK2 protein, mouse
  • G1p2 protein, mouse
  • GTPase-Activating Proteins
  • Guanine Nucleotide Exchange Factors
  • Ifit1 protein, mouse
  • Interferon-alpha
  • Membrane Glycoproteins
  • Neuropeptides
  • Pik3ap1 protein, mouse
  • RNA-Binding Proteins
  • Rac1 protein, mouse
  • Tlr7 protein, mouse
  • Tlr9 protein, mouse
  • Toll-Like Receptor 7
  • Toll-Like Receptor 9
  • Ubiquitins
  • rac1 GTP-Binding Protein