A CCR5+ memory subset within HIV-1-infected primary resting CD4+ T cells is permissive for replication-competent, latently infected viruses in vitro

BMC Res Notes. 2019 Apr 29;12(1):242. doi: 10.1186/s13104-019-4281-5.

Abstract

Objective: Resting CD4+ T cells are major reservoirs of latent HIV-1 infection, and may be formed during the early phase of the infection. Although CCR5-tropic (R5) HIV-1 is highly transmissible during the early phase, newly infected individuals have usually been exposed to a mixture of R5 and CXCR4-tropic (X4) viruses, and X4 viral DNA is also detectable in the host. Our aim was to identify which subsets of resting CD4+ T cells contribute to forming the latent reservoir in the presence of both X4 and R5 viruses.

Results: Primary resting CD4+ naïve T (TN) cells, CCR5- memory T (TM) cells, and CCR5+ TM cells isolated by flow cytometry were infected simultaneously with X4 and R5 HIV-1, which harbored different reporter genes, and were cultured in the resting condition. Flow cytometry at 3 days post-infection demonstrated that X4 HIV-1+ cells were present in all three subsets of cells, whereas R5 HIV-1+ cells were present preferentially in CCR5+ TM cells, but not in TN cells. Following CD3/CD28-mediated activation at 3 days post-infection, numbers of R5 HIV-1+ cells and X4 HIV-1+ cells increased significantly only in the CCR5+ TM subset, suggesting that it provides a major reservoir of replication-competent, latently infected viruses.

Keywords: HIV; Latent reservoir; Resting CD4+ T cells.

MeSH terms

  • Adult
  • CD4-Positive T-Lymphocytes / immunology
  • CD4-Positive T-Lymphocytes / virology*
  • DNA, Viral / genetics*
  • DNA, Viral / metabolism
  • Gene Expression
  • HIV-1 / pathogenicity
  • HIV-1 / physiology*
  • Healthy Volunteers
  • Host-Pathogen Interactions / genetics
  • Host-Pathogen Interactions / immunology
  • Humans
  • Immunologic Memory
  • Primary Cell Culture
  • Receptors, CCR5 / genetics*
  • Receptors, CCR5 / immunology
  • Receptors, CXCR4 / genetics*
  • Receptors, CXCR4 / immunology
  • Viral Tropism
  • Virus Latency*
  • Virus Replication

Substances

  • CCR5 protein, human
  • CXCR4 protein, human
  • DNA, Viral
  • Receptors, CCR5
  • Receptors, CXCR4