miR-9-5p Suppresses Malignant Biological Behaviors of Human Gastric Cancer Cells by Negative Regulation of TNFAIP8L3

Dig Dis Sci. 2019 Oct;64(10):2823-2829. doi: 10.1007/s10620-019-05626-2. Epub 2019 May 28.

Abstract

Background: MicroRNA is essential for the malignant progression of human gastric cancer (GC), which is a leading cause of cancer deaths. However, the mechanism is still not so clear.

Aims: In our present research, we investigated the effect of miR-9-5p in GC.

Methods: We detected miR-9-5p expression in human gastric epithelial cell (GES-1) and GC cells (AGS, BGC-823, MKN-45, and MGC-803), plasma of normal or GC patients, as well as orthotopic xenograft mouse models by real-time PCR. The migration ability was detected by Transwell assays after miR-9-5p mimic or inhibitor transfection in GC cells.

Results: Our results showed that miR-9-5p expression in GC cells and plasma was significantly decreased. miR-9-5p inhibited migration of GC cells by regulating TNFAIP8L3 directly. Low expression of miR-9-5p in GC patients hardly suppressed the migration mediated by TNFAIP8L3.

Conclusions: miR-9-5p, as a potential tumor suppressor gene, is closely related to the malignant progression of GC. Exploring the regulation between miR-9-5p and TNFAIP8L3 may provide a novel strategy for GC treatment.

Keywords: 3′UTR; Gastric cancer; Migration; TNFAIP8L3; miR-9-5p.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Cell Line, Tumor
  • Gene Expression Regulation, Neoplastic
  • Genes, Tumor Suppressor
  • Humans
  • Intracellular Signaling Peptides and Proteins / metabolism*
  • Mice
  • MicroRNAs / metabolism*
  • Stomach Neoplasms* / metabolism
  • Stomach Neoplasms* / pathology
  • Tumor Necrosis Factor-alpha / metabolism
  • Xenograft Model Antitumor Assays / methods

Substances

  • Intracellular Signaling Peptides and Proteins
  • MIRN9 microRNA, mouse
  • MIRN92 microRNA, human
  • MicroRNAs
  • TNFAIP8L3 protein, human
  • Tumor Necrosis Factor-alpha