Regulation of interleukin 2 and interleukin 2 receptor gene expression in human T cells: I. Effect of Ca2+-ionophore on phorbol myristate acetate co-stimulated cells

Clin Exp Immunol. 1987 Aug;69(2):433-40.

Abstract

Human peripheral blood T lymphocytes were treated with phorbol myristate acetate (PMA), an activator of protein kinase C (PKC) activity, and with the calcium ionophore A23187. The resulting accumulation of specific mRNA for interleukin 2 (IL-2) and interleukin 2 receptor (IL-2R), as well as IL-2 secretion and membrane IL-2R expression were examined. At low concentrations (0.1 microM), A23187 synergized maximally with PMA to induce proliferation, to increase IL-2R mRNA levels and the expression of membrane IL-2R, and to produce a low but sufficient accumulation of IL-2 mRNA and IL-2 secretion. A high concentration of A23187 (1.0 microM) did not show any synergism for the accumulation of IL-2R mRNA, membrane IL-2R expression and inhibited the proliferation of PMA co-stimulated T cells. It did, however, induce maximum accumulation of IL-2 mRNA and IL-2 secretion.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Calcimycin / pharmacology*
  • Cell Division / drug effects
  • Drug Synergism
  • Gene Expression Regulation / drug effects*
  • Humans
  • Interleukin-2 / biosynthesis
  • Interleukin-2 / genetics*
  • Lymphocyte Activation / drug effects*
  • RNA, Messenger / analysis
  • Receptors, Immunologic / analysis
  • Receptors, Immunologic / genetics*
  • Receptors, Interleukin-2
  • T-Lymphocytes / immunology
  • Tetradecanoylphorbol Acetate / pharmacology

Substances

  • Interleukin-2
  • RNA, Messenger
  • Receptors, Immunologic
  • Receptors, Interleukin-2
  • Calcimycin
  • Tetradecanoylphorbol Acetate