Small airway inflammation is associated with residual airway hyperresponsiveness in Th2-high asthma

J Asthma. 2020 Sep;57(9):933-941. doi: 10.1080/02770903.2019.1628251. Epub 2019 Jun 19.

Abstract

Background: Asthma is characterized by airway inflammation, variable airflow obstruction, and airway hyperresponsiveness (AHR). Generally, AHR takes longer to resolve than does airflow obstruction or clinical symptoms. AHR occasionally persists despite adequate asthma treatment.Objective: To evaluate factors which associates with residual AHR in patients with seemingly remitted airway inflammation.Methods: Patients who exhibited high fractional exhaled nitric oxide (FeNO) levels (>25 ppb) at the first visit (Visit 1) and normalized FeNO levels (<25 ppb) after adequate asthma treatment, including inhaled corticosteroid administration (Visit 2), were analyzed. Patients underwent a blood test, FeNO and small airway/alveolar nitric oxide concentration (CANO) measurements and a methacholine challenge test (continuous inhalation method) at both visits. Clinical indices were compared between patients with and without residual AHR.Results: Fifty patients were analyzed. All exhibited high FeNO levels at Visit 1 [mean, 54.0 ppb (95% confidence interval, 42.4-65.5)] and improvement of FeNO levels at Visit 2 [20.4 (19.2-21.6)] (p < 0.0001). Thirty-three patients (66%) had remission of AHR at Visit 2. No significant differences were observed between patients with and without residual AHR in terms of FeNO levels, lung function parameters and blood eosinophil counts at both visits. CANO level at Visit 2 was the only factor that significantly differed between patients with residual AHR [2.7 (1.9-3.6)] and those who achieved AHR remission [0.8 (0.5-1.0)] (p < 0.0001).Conclusion: Small airway inflammation, as assessed by CANO, was associated with residual AHR in patients with Th2-high asthma.

Keywords: Biomarkers; Control/Management; airway hyperresponsiveness; alveolar nitric oxide; exhaled nitric oxide.

Publication types

  • Observational Study

MeSH terms

  • Administration, Inhalation
  • Asthma / blood
  • Asthma / drug therapy
  • Asthma / immunology*
  • Breath Tests
  • Bronchial Provocation Tests / methods
  • CD4 Lymphocyte Count
  • Female
  • Glucocorticoids / therapeutic use
  • Humans
  • Male
  • Methacholine Chloride / administration & dosage
  • Nitric Oxide / analysis
  • Respiratory Hypersensitivity / blood
  • Respiratory Hypersensitivity / diagnosis*
  • Respiratory Hypersensitivity / immunology
  • Spirometry
  • Th2 Cells / immunology*

Substances

  • Glucocorticoids
  • Methacholine Chloride
  • Nitric Oxide