Display of malaria transmission-blocking antigens on chimeric duck hepatitis B virus-derived virus-like particles produced in Hansenula polymorpha

PLoS One. 2019 Sep 4;14(9):e0221394. doi: 10.1371/journal.pone.0221394. eCollection 2019.

Abstract

Background: Malaria caused by Plasmodium falciparum is one of the major threats to human health globally. Despite huge efforts in malaria control and eradication, highly effective vaccines are urgently needed, including vaccines that can block malaria transmission. Chimeric virus-like particles (VLP) have emerged as a promising strategy to develop new malaria vaccine candidates.

Methods: We developed yeast cell lines and processes for the expression of malaria transmission-blocking vaccine candidates Pfs25 and Pfs230 as VLP and VLP were analyzed for purity, size, protein incorporation rate and expression of malaria antigens.

Results: In this study, a novel platform for the display of Plasmodium falciparum antigens on chimeric VLP is presented. Leading transmission-blocking vaccine candidates Pfs25 and Pfs230 were genetically fused to the small surface protein (dS) of the duck hepatitis B virus (DHBV). The resulting fusion proteins were co-expressed in recombinant Hansenula polymorpha (syn. Pichia angusta, Ogataea polymorpha) strains along with the wild-type dS as the VLP scaffold protein. Through this strategy, chimeric VLP containing Pfs25 or the Pfs230-derived fragments Pfs230c or Pfs230D1M were purified. Up to 100 mg chimeric VLP were isolated from 100 g dry cell weight with a maximum protein purity of 90% on the protein level. Expression of the Pfs230D1M construct was more efficient than Pfs230c and enabled VLP with higher purity. VLP showed reactivity with transmission-blocking antibodies and supported the surface display of the malaria antigens on the native VLP.

Conclusion: The incorporation of leading Plasmodium falciparum transmission-blocking antigens into the dS-based VLP scaffold is a promising novel strategy for their display on nano-scaled particles. Competitive processes for efficient production and purification were established in this study.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Antibodies, Blocking / immunology
  • Antigens, Protozoan / genetics
  • Antigens, Protozoan / metabolism*
  • Ducks / virology
  • Hepatitis B Virus, Duck / genetics*
  • Humans
  • Malaria / prevention & control
  • Malaria Vaccines / biosynthesis*
  • Malaria Vaccines / immunology
  • Malaria Vaccines / isolation & purification
  • Pichia / metabolism*
  • Plasmodium falciparum / metabolism
  • Recombinant Fusion Proteins / biosynthesis
  • Recombinant Fusion Proteins / immunology
  • Recombinant Fusion Proteins / isolation & purification
  • Vaccines, Virus-Like Particle / biosynthesis*
  • Vaccines, Virus-Like Particle / immunology
  • Vaccines, Virus-Like Particle / isolation & purification

Substances

  • Antibodies, Blocking
  • Antigens, Protozoan
  • Malaria Vaccines
  • Recombinant Fusion Proteins
  • Vaccines, Virus-Like Particle

Grants and funding

This work was supported by PATH Malaria Vaccine Initiative; National Health and Medical Research Council of Australia (Senior Research Fellowship and Program Grant to JGB). Burnet Institute is supported by funding from the NHMRC Independent Research Institutes Infrastructure Support Scheme and a Victorian State Government Operational Infrastructure grant. ARTES Biotechnology GmbH provided support in the form of salaries for authors DW, MS, MW, VJ and MP. Juliane Merz is employed by Evonik Technology & Infrastructure GmbH. Evonik Technology & Infrastructure GmbH provided support in the form of salary for author JM, but did not have any additional role in the study design, data collection and analysis, or preparation of the manuscript. Evonik Technology & Infrastructure GmbH expressly agreed on publishing the manuscript. The specific roles of JM are articulated in the ‘author contributions’ section.