Failure of percutaneous transluminal coronary angioplasty to stimulate platelet and prostaglandin activity

Cathet Cardiovasc Diagn. 1985;11(3):247-54. doi: 10.1002/ccd.1810110304.

Abstract

Platelet function and prostaglandin activity were evaluated in nine patients with coronary artery disease undergoing percutaneous left anterior descending coronary artery angioplasty (PTCA) and compared to nine normal controls. Transcoronary measurements (arterial-coronary sinus) of platelet counts, mean platelet volume, platelet factor 4 (PF4), beta thromboglobulin, thromboxane (B2), and 6-keto-PGF 1 alpha were made. When compared to normal controls, the patients with coronary artery disease had higher circulating baseline levels of PF4 in the coronary sinus. There was no transcardiac production of any factor at baseline or immediately after infusion of nitroglycerin or performance of PTCA. These results suggest that PTCA does not grossly alter arachidonic acid metabolism or platelet activity.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • 6-Ketoprostaglandin F1 alpha / blood
  • Adult
  • Aged
  • Angioplasty, Balloon*
  • Cardiac Catheterization
  • Coronary Disease / blood
  • Coronary Disease / therapy*
  • Humans
  • Middle Aged
  • Platelet Aggregation*
  • Platelet Count
  • Platelet Factor 4 / physiology
  • Prostaglandins / blood*
  • Thromboxane B2 / blood
  • beta-Thromboglobulin / metabolism

Substances

  • Prostaglandins
  • beta-Thromboglobulin
  • Platelet Factor 4
  • Thromboxane B2
  • 6-Ketoprostaglandin F1 alpha