Induction to erythroid differentiation of K562 cells by 1-beta-D-arabinofuranosylcytosine is inhibited by iron chelators: reversion by treatment with hemin

Blut. 1988 Jul;57(1):25-30. doi: 10.1007/BF00320631.

Abstract

Erythroid differentiation of human leukemic K 562 cells is inhibited by the iron chelator desferrioxamine (DF). In addition, desferrioxamine induces an increase of uptake of hemin. When hemin is added to the culture medium, the DF-mediated inhibitory effects on erythroid induction are reversed. Briefly, hemin allows hemoglobin synthesis by K 562 cells induced to erythroid differentiation by 1-beta-D-arabinofuranosylcytosine (ara-C) and treated with 12.5 micrograms/ml DF. In addition, it was found that hemin treatment leads to a reversion of inhibition of K 562 cell proliferation mediated by 50-75 micrograms/ml DF. This effect of hemin was also detected in other cultured human tumor cell lines (B-lymphoid, erythroleukemic and from breast carcinomas, melanomas and kidney carcinomas).

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Cell Differentiation / drug effects
  • Cell Line
  • Cytarabine / pharmacology*
  • Deferoxamine / pharmacology*
  • Erythropoiesis / drug effects*
  • Growth Inhibitors / pharmacology*
  • Heme / analogs & derivatives*
  • Hemin / pharmacology*
  • Hemoglobins / metabolism
  • Humans
  • Kinetics
  • Leukemia, Erythroblastic, Acute / metabolism
  • Leukemia, Erythroblastic, Acute / pathology*

Substances

  • Growth Inhibitors
  • Hemoglobins
  • Cytarabine
  • Heme
  • Hemin
  • Deferoxamine