The largest caucasian kindred with dentatorubral-pallidoluysian atrophy: A founder mutation in italy

Mov Disord. 2019 Dec;34(12):1919-1924. doi: 10.1002/mds.27879. Epub 2019 Nov 21.

Abstract

Background: Dentatorubral-pallidoluysian atrophy is a hereditary neurodegenerative disease prevalently reported in Japan but rare in Caucasians. The objective of this study was to reconstruct the pedigree of Italian dentatorubral-pallidoluysian atrophy familial cases describing their clinical features.

Methods: We investigated 6 apparently unrelated dentatorubral-pallidoluysian atrophy families comprising a total of 51 affected individuals: 13 patients were clinically examined, and for 38 patients clinical data were collected from clinical sources. The dentatorubral-pallidoluysian atrophy diagnosis was genetically confirmed in 18 patients. Genealogical data from historical archives were analyzed.

Results: All 6 families were unified in a large pedigree deriving from a founder couple originating from Monte San Giuliano (Italy) in the late 1500s, with 51 affected subjects over the last 4 generations. Wide phenotypical variability in age at onset and clinical features was confirmed. Epilepsy was more frequent in juvenile cases than in late adults, with cognitive/psychiatric and motor disorders observed regardless of age at onset.

Conclusions: We have described the largest Caucasian dentatorubral-pallidoluysian atrophy pedigree from a single founder couple. The introduction of the dentatorubral-pallidoluysian atrophy gene in Italy could have arisen as a result of trade relationships between the Spanish or Portuguese and the Japanese in the 1500s. © 2019 International Parkinson and Movement Disorder Society.

Keywords: ATN1 gene; cerebellar cognitive-affective syndrome; dentatorubral-pallidoluysian atrophy; founder effect; genealogical method.

MeSH terms

  • Adolescent
  • Adult
  • Aged
  • Child
  • Epilepsy / complications
  • Epilepsy / epidemiology
  • Family
  • Female
  • Founder Effect*
  • Humans
  • Italy / epidemiology
  • Male
  • Middle Aged
  • Mutation / genetics*
  • Myoclonic Epilepsies, Progressive / epidemiology*
  • Myoclonic Epilepsies, Progressive / genetics*
  • Myoclonic Epilepsies, Progressive / psychology
  • Neuropsychological Tests
  • Pedigree
  • Trinucleotide Repeats
  • White People
  • Young Adult