HBeAg Negativity Is Associated With More Advanced Liver Fibrosis in Patients With Chronic Hepatitis B: A Propensity Score-Matching Analysis

J Clin Gastroenterol. 2020 Oct;54(9):826-831. doi: 10.1097/MCG.0000000000001291.

Abstract

Background: Serum hepatitis B e antigen (HBeAg) status is associated with the progression of chronic hepatitis B (CHB). The authors aimed to investigate the relationship between HBeAg status and liver pathology in CHB patients.

Methods: A total of 683 treatment-naive CHB patients who had undergone liver biopsy were retrospectively enrolled from 2 medical centers. Propensity score-matching (PSM) method was performed to adjust the imbalance of baseline confounders between HBeAg-positive and HBeAg-negative CHB patients.

Results: HBeAg-negative CHB patients (n=338) exhibited more advanced liver fibrosis than HBeAg-positive CHB patients (n=345) before PSM (P<0.001). However, there were no significant differences in the distribution of inflammation grades between HBeAg-positive and HBeAg-negative CHB patients (P=0.051). Of these 683 CHB patients, 123 patients were included in each group after PSM. HBeAg-negative CHB patients still showed significantly advanced liver fibrosis as compared with HBeAg-positive CHB patients (P=0.03) after PSM. Furthermore, the distribution of liver inflammation grades in the HBeAg-negative CHB patients was also more severe than patients with HBeAg-positive (P=0.037). HBeAg-negative status was identified as an independent risk factor of significant liver fibrosis (P=0.011) by multivariate analysis.

Conclusions: HBeAg negativity is associated with more advanced liver fibrosis in CHB patients.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • DNA, Viral
  • Hepatitis B e Antigens*
  • Hepatitis B virus / genetics
  • Hepatitis B, Chronic* / complications
  • Hepatitis B, Chronic* / pathology
  • Humans
  • Liver / pathology
  • Liver Cirrhosis / pathology
  • Propensity Score
  • Retrospective Studies

Substances

  • DNA, Viral
  • Hepatitis B e Antigens