Practical roles for molecular diagnostic testing in ovarian adult granulosa cell tumour, Sertoli-Leydig cell tumour, microcystic stromal tumour and their mimics

Histopathology. 2020 Jan;76(1):11-24. doi: 10.1111/his.13978.

Abstract

Within the last decade, molecular advances have provided insights into the genetics of several ovarian sex cord-stromal tumours that have otherwise been enigmatic. Chief among these advances are the identification of FOXL2, DICER1 and CTNNB1 mutations in adult granulosa cell tumours, Sertoli-Leydig cell tumours (SLCTs), and microcystic stromal tumours (MCSTs), respectively. As access to molecular diagnostic laboratories continues to become more widely available, the potential roles for tumour mutation testing in the pathological diagnosis of these tumours merit discussion. Furthermore, links to inherited cancer susceptibility syndromes may exist for some women with SLCT (DICER1 syndrome) and MCST [familial adenomatous polyposis (FAP)]. This review will address practical issues in deciding when and how to apply mutation testing in the diagnosis of these three sex cord-stromal tumours. The pathologist's role in recommending referral for formal risk assessment for DICER1 syndrome and FAP will also be discussed.

Keywords: CTNNB1; DICER1; FOXL2; Sertoli-Leydig cell tumour; granulosa cell tumour; microcystic stromal tumour.

Publication types

  • Review

MeSH terms

  • Diagnosis, Differential
  • Endometrial Stromal Tumors / diagnosis*
  • Endometrial Stromal Tumors / genetics
  • Endometrial Stromal Tumors / pathology
  • Female
  • Granulosa Cell Tumor / diagnosis*
  • Granulosa Cell Tumor / genetics
  • Granulosa Cell Tumor / pathology
  • Humans
  • Ovarian Neoplasms / diagnosis*
  • Ovarian Neoplasms / genetics
  • Ovarian Neoplasms / pathology
  • Pathology, Molecular
  • Sertoli-Leydig Cell Tumor / diagnosis*
  • Sertoli-Leydig Cell Tumor / genetics
  • Sertoli-Leydig Cell Tumor / pathology