Immune Consequences of Endothelial Cells' Activation and Dysfunction During Sepsis

Crit Care Clin. 2020 Apr;36(2):401-413. doi: 10.1016/j.ccc.2019.12.001. Epub 2020 Jan 30.

Abstract

The vascular endothelium provides a direct interface between circulating blood cells and parenchymal cells. Thus, it has a key role in vasomotor tone regulation, primary hemostasis, vascular barrier, and immunity. In the case of systemic inflammation, endothelial cell (EC) activation initiates a powerful innate immune response to eliminate the pathogen. In some specific conditions, ECs may also contribute to the activation of adaptive immunity and the recruitment of antigen-specific lymphocytes. However, the loss of EC functions or an exaggerated activation of ECs during sepsis can lead to multiorgan failure.

Keywords: Adaptive immunity; Barrier function; Endothelial dysfunction; Hemostasis; Innate immunity; Sepsis.

Publication types

  • Review

MeSH terms

  • Adaptive Immunity
  • Endothelial Cells / immunology*
  • Endothelium, Vascular / immunology*
  • Humans
  • Immunity, Innate
  • Multiple Organ Failure / immunology
  • Sepsis / immunology*