4E-BP-Dependent Translational Control of Irf8 Mediates Adipose Tissue Macrophage Inflammatory Response

J Immunol. 2020 May 1;204(9):2392-2400. doi: 10.4049/jimmunol.1900538. Epub 2020 Mar 25.

Abstract

Deregulation of mRNA translation engenders many human disorders, including obesity, neurodegenerative diseases, and cancer, and is associated with pathogen infections. The role of eIF4E-dependent translational control in macrophage inflammatory responses in vivo is largely unexplored. In this study, we investigated the involvement of the translation inhibitors eIF4E-binding proteins (4E-BPs) in the regulation of macrophage inflammatory responses in vitro and in vivo. We show that the lack of 4E-BPs exacerbates inflammatory polarization of bone marrow-derived macrophages and that 4E-BP-null adipose tissue macrophages display enhanced inflammatory gene expression following exposure to a high-fat diet (HFD). The exaggerated inflammatory response in HFD-fed 4E-BP-null mice coincides with significantly higher weight gain, higher Irf8 mRNA translation, and increased expression of IRF8 in adipose tissue compared with wild-type mice. Thus, 4E-BP-dependent translational control limits, in part, the proinflammatory response during HFD. These data underscore the activity of the 4E-BP-IRF8 axis as a paramount regulatory mechanism of proinflammatory responses in adipose tissue macrophages.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adaptor Proteins, Signal Transducing / genetics*
  • Adipose Tissue / metabolism*
  • Animals
  • Bone Marrow / metabolism
  • Diet, High-Fat / methods
  • Eukaryotic Initiation Factor-4E / genetics
  • Gene Expression / genetics
  • Inflammation / genetics*
  • Inflammation / metabolism
  • Interferon Regulatory Factors / genetics*
  • Macrophages / metabolism*
  • Male
  • Mice
  • Mice, Inbred C57BL
  • Mice, Knockout
  • Protein Biosynthesis / genetics*

Substances

  • Adaptor Proteins, Signal Transducing
  • Eukaryotic Initiation Factor-4E
  • Interferon Regulatory Factors
  • interferon regulatory factor-8