Structural Basis for the Activation and Target Site Specificity of CDC7 Kinase

Structure. 2020 Aug 4;28(8):954-962.e4. doi: 10.1016/j.str.2020.05.010. Epub 2020 Jun 9.

Abstract

CDC7 is an essential Ser/Thr kinase that acts upon the replicative helicase throughout the S phase of the cell cycle and is activated by DBF4. Here, we present crystal structures of a highly active human CDC7-DBF4 construct. The structures reveal a zinc-finger domain at the end of the kinase insert 2 that pins the CDC7 activation loop to motif M of DBF4 and the C lobe of CDC7. These interactions lead to ordering of the substrate-binding platform and full opening of the kinase active site. In a co-crystal structure with a mimic of MCM2 Ser40 phosphorylation target, the invariant CDC7 residues Arg373 and Arg380 engage phospho-Ser41 at substrate P+1 position, explaining the selectivity of the S-phase kinase for Ser/Thr residues followed by a pre-phosphorylated or an acidic residue. Our results clarify the role of DBF4 in activation of CDC7 and elucidate the structural basis for recognition of its preferred substrates.

Keywords: CDC7; DBF4; DDK; XL413; bisubstrate; cell cycle; crystal structure; kinase; kinase inhibitor; zinc-binding domain.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Catalytic Domain
  • Cell Cycle Proteins / chemistry*
  • Cell Cycle Proteins / metabolism
  • Humans
  • Molecular Docking Simulation
  • Phosphorylation
  • Protein Binding
  • Protein Serine-Threonine Kinases / chemistry*
  • Protein Serine-Threonine Kinases / metabolism
  • Substrate Specificity
  • Zinc Fingers

Substances

  • Cell Cycle Proteins
  • DBF4 protein, human
  • CDC7 protein, human
  • Protein Serine-Threonine Kinases