Histological diagnosis of immune checkpoint inhibitor induced acute renal injury in patients with metastatic melanoma: a retrospective case series report

BMC Nephrol. 2020 Sep 7;21(1):391. doi: 10.1186/s12882-020-02044-9.

Abstract

Background: Immune checkpoint inhibitors (ICI) have become the standard of care in many oncological conditions but are associated with a spectrum of renal immune-related adverse events (IrAEs). We aimed to describe the spectrum, histology, management and outcomes of renal IrAE in patients with metastatic melanoma undergoing ICI therapy.

Methods: We conducted a retrospective review of 23 patients with a diagnosis of metastatic melanoma treated with ICI between January 2017 and April 2019 who developed a renal IrAE. Baseline demographic data, biochemical and histopathological results, management and outcomes were analyzed.

Results: The majority of patients who developed renal irAE were male and received combination immunotherapy. The median time of onset from initiation of ICI therapy to renal IrAE was 4 months. 52% of the treated renal IrAE had histopathologically confirmed renal IrAE. The most common histological pattern of injury was acute tubulo-interstitial nephritis (92%). One patient developed anti-GBM disease with non-dialysis dependent stage 5 CKD. In tubulointerstitial injury, there was no association between peak creatinine, renal recovery and histologically reported inflammation or fibrosis. Patients with renal IrAE demonstrated persisting renal dysfunction at 3, 6 and 12 months with a mean baseline, 3 and 12 month creatinine of 90.0 μmol/L, 127.0 μmol/L and 107.5 μmol/L respectively.

Conclusion: Renal IrAE is most commonly attributable to steroid responsive acute tubulointerstitial nephritis. The outcome of rarer pathologies such as anti-GBM disease may be adversely affected by a delayed diagnosis. There is persisting renal dysfunction following an episode of renal IrAE that may have impact on future renal and overall survival outcomes.

Keywords: AKI; Glomerulonephritis; Immune checkpoint inhibitor; Immunology; Renal biopsy; Tubulo interstitial nephritis.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Acute Kidney Injury / chemically induced*
  • Acute Kidney Injury / pathology
  • Adult
  • Aged
  • Aged, 80 and over
  • Anti-Glomerular Basement Membrane Disease / chemically induced
  • Anti-Glomerular Basement Membrane Disease / pathology
  • Antibodies, Monoclonal, Humanized / adverse effects
  • Female
  • Humans
  • Immune Checkpoint Inhibitors / adverse effects*
  • Ipilimumab / adverse effects
  • Male
  • Melanoma / drug therapy*
  • Melanoma / secondary
  • Middle Aged
  • Nephritis, Interstitial / chemically induced*
  • Nephritis, Interstitial / pathology
  • Nivolumab / adverse effects
  • Renal Insufficiency, Chronic / chemically induced
  • Renal Insufficiency, Chronic / pathology
  • Retrospective Studies
  • Skin Neoplasms / drug therapy*
  • Skin Neoplasms / pathology

Substances

  • Antibodies, Monoclonal, Humanized
  • Immune Checkpoint Inhibitors
  • Ipilimumab
  • Nivolumab
  • pembrolizumab

Supplementary concepts

  • Acute Tubulointerstitial Nephritis