Predictive value of tumor-infiltrating lymphocytes for pathological response to neoadjuvant chemotherapy in breast cancer patients with axillary lymph node metastasis

Surg Today. 2021 Apr;51(4):595-604. doi: 10.1007/s00595-020-02157-6. Epub 2020 Oct 14.

Abstract

Purpose: We investigated the role of tumor-infiltrating lymphocytes (TILs) in pretreatment primary breast cancer to predict pathological response to neoadjuvant chemotherapy (NAC) in patients with clinical node-positive disease (cN +).

Methods: The subjects of this study were 60 patients with cN + , who received NAC followed by breast surgery with axillary lymph node dissection (ALND). We conducted a semi-quantitative assessment of TILs in pretreatment primary tumors and their association with clinicopathological factors and axillary lymph node metastasis.

Results: We observed a higher number of TILs in tumors with negative hormone receptors, positive human epidermal growth factor receptor 2, or high Ki67. TILs were associated with a favorable response to NAC in primary tumors. The rate of axillary pathologic complete response (Ax-pCR) was significantly higher in patients with a high number of TILs than in patients with a low number of TILs (72.0% versus 17.1%, p < 0.001). In multivariable analysis, a high number of TILs was a significant predictor of Ax-pCR as well as of pCR of the primary tumor after NAC. Importantly, all patients with HER2-positive tumors in the high TILs group showed Ax-pCR on ALND.

Conclusion: TILs in pretreatment primary breast cancer had the potential to predict therapeutic efficacy of NAC in patients with clinical node-positive disease.

Keywords: Axillary dissection; Axillary pathological complete response; Breast cancer; Neoadjuvant chemotherapy; Tumor-infiltrating lymphocytes.

MeSH terms

  • Axilla
  • Breast Neoplasms / drug therapy*
  • Breast Neoplasms / metabolism
  • Breast Neoplasms / pathology*
  • Female
  • Forecasting
  • Humans
  • Ki-67 Antigen / metabolism
  • Lymph Nodes / pathology*
  • Lymphatic Metastasis / pathology*
  • Lymphocytes, Tumor-Infiltrating / metabolism
  • Lymphocytes, Tumor-Infiltrating / pathology*
  • Neoadjuvant Therapy*
  • Receptor, ErbB-2 / metabolism
  • Treatment Outcome

Substances

  • Ki-67 Antigen
  • MKI67 protein, human
  • ERBB2 protein, human
  • Receptor, ErbB-2