Cardiac β-Adrenergic Receptor Downregulation, Evaluated by Cardiac PET, in Chronotropic Incompetence

J Nucl Med. 2021 Jul 1;62(7):996-998. doi: 10.2967/jnumed.120.253419. Epub 2020 Dec 18.

Abstract

The mechanism of chronotropic incompetence (CTI), which has been associated with autonomic dysfunction, has not been elucidated in patients without heart failure (HF). Methods: Cardiac PET using 11C-CGP12177 was performed to investigate the cardiac β-adrenergic receptor density (β-ARD) in 13 patients with CTI without HF and 6 healthy controls. The maximum number of available specific 11C-CGP12177 binding sites per gram of tissue was calculated in regions of interest using an established graphical method. Results: Peak heart rate was significantly lower in CTI patients than in controls (116.9 ± 11.0 vs. 154.8 ± 14.4 beats/min, P < 0.001). β-ARD of the total myocardium was significantly lower in CTI patients than in controls (4.3 ± 1.7 vs. 7.0 ± 1.7 pmol/mL, P = 0.005). Conclusion: β-adrenergic receptor downregulation was demonstrated in patients with CTI without HF. Decreased β-ARD is a common feature in patients with CTI, with or without HF.

Keywords: PET; chronotropic incompetence; sympathetic nervous system; β-adrenergic receptor.

MeSH terms

  • Adult
  • Aged
  • Down-Regulation*
  • Female
  • Heart / diagnostic imaging
  • Heart Rate*
  • Humans
  • Male
  • Middle Aged
  • Myocardium / metabolism
  • Positron-Emission Tomography*
  • Propanolamines
  • Receptors, Adrenergic, beta* / metabolism

Substances

  • Receptors, Adrenergic, beta
  • CGP 12177
  • Propanolamines