Arterial stiffness in acute COVID-19 and potential associations with clinical outcome

J Intern Med. 2021 Aug;290(2):437-443. doi: 10.1111/joim.13275. Epub 2021 Mar 21.

Abstract

Background: Coronavirus disease 2019 (COVID-19) interferes with the vascular endothelium. It is not known whether COVID-19 additionally affects arterial stiffness.

Methods: This case-control study compared brachial-ankle pulse wave (baPWV) and carotid-femoral pulse wave velocities (cfPWV) of acutely ill patients with and without COVID-19.

Results: Twenty-two COVID-19 patients (50% females, 77 [67-84] years) were compared with 22 age- and sex-matched controls. In COVID-19 patients, baPWV (19.9 [18.4-21.0] vs. 16.0 [14.2-20.4], P = 0.02) and cfPWV (14.3 [13.4-16.0] vs. 11.0 [9.5-14.6], P = 0.01) were higher than in the controls. In multiple regression analysis, COVID-19 was independently associated with higher cfPWV (β = 3.164, P = 0.004) and baPWV (β = 3.532, P = 0.003). PWV values were higher in nonsurvivors. In survivors, PWV correlated with length of hospital stay.

Conclusion: COVID-19 appears to be related to an enhanced PWV reflecting an increase in arterial stiffness. Higher PWV might be related to an increased length of hospital stay and mortality.

Keywords: COVID-19; SARS-CoV-2; arterial stiffness; cardiovascular risk; pulse wave velocity.

MeSH terms

  • Aged
  • Aged, 80 and over
  • Brachial Artery / physiopathology
  • COVID-19 / mortality*
  • COVID-19 / physiopathology*
  • Carotid Arteries / physiopathology
  • Case-Control Studies
  • Female
  • Femoral Artery / physiopathology
  • Humans
  • Length of Stay
  • Male
  • Pulse Wave Analysis
  • Survivors
  • Vascular Stiffness / physiology*