PIM2-mediated phosphorylation contributes to granulosa cell survival via resisting apoptosis during folliculogenesis

Clin Transl Med. 2021 Mar;11(3):e359. doi: 10.1002/ctm2.359.

Authors

Lei Wang¹, Yaru Chen¹, Shang Wu¹, Ling Wang¹, Feng Tan¹, Fenge Li^{1

2}

Affiliations

¹ Key Laboratory of Swine Genetics and Breeding of Ministry of Agriculture and Rural Affairs & Key Laboratory of Agricultural Animal Genetics, Breeding and Reproduction of Ministry of Education, Huazhong Agricultural University, Wuhan, China.
² The Cooperative Innovation Center for Sustainable Pig Production, Wuhan, China.

No abstract available

Publication types

Letter
Research Support, Non-U.S. Gov't

MeSH terms

Animals
Apoptosis / genetics
Disease Models, Animal
Female
Granulosa Cells / metabolism*
Mice
Mice, Inbred C57BL
Phosphorylation / genetics
Primary Ovarian Insufficiency / genetics*
Primary Ovarian Insufficiency / metabolism*
Protein Serine-Threonine Kinases / genetics*
Protein Serine-Threonine Kinases / metabolism*
Proto-Oncogene Proteins / genetics*
Proto-Oncogene Proteins / metabolism*

Substances

Pim2 protein, mouse
Proto-Oncogene Proteins
Protein Serine-Threonine Kinases