Pathology, Radiology, and Genetics of Interstitial Lung Disease in Patients With Shortened Telomeres

Am J Surg Pathol. 2021 Jul 1;45(7):871-884. doi: 10.1097/PAS.0000000000001725.

Abstract

Interstitial lung diseases (ILDs) in patients with shortened telomeres have not been well characterized. We describe demographic, radiologic, histopathologic, and molecular features, and p16 expression in patients with telomeres ≤10th percentile (shortened telomeres) and compare them to patients with telomere length >10th percentile. Lung explants, wedge biopsies, and autopsy specimens of patients with telomere testing were reviewed independently by 3 pathologists using defined parameters. High-resolution computed tomography scans were reviewed by 3 radiologists. p16-positive fibroblast foci were quantified. A multidisciplinary diagnosis was recorded. Patients with shortened telomeres (N=26) were morphologically diagnosed as usual interstitial pneumonia (UIP) (N=11, 42.3%), chronic hypersensitivity pneumonitis (N=6, 23.1%), pleuroparenchymal fibroelastosis, fibrotic nonspecific interstitial pneumonia, desquamative interstitial pneumonia (N=1, 3.8%, each), and fibrotic interstitial lung disease (fILD), not otherwise specified (N=6, 23.1%). Patients with telomeres >10th percentile (N=18) showed morphologic features of UIP (N=9, 50%), chronic hypersensitivity pneumonitis (N=3, 16.7%), fibrotic nonspecific interstitial pneumonia (N=2, 11.1%), or fILD, not otherwise specified (N=4, 22.2%). Patients with shortened telomeres had more p16-positive foci (P=0.04). The number of p16-positive foci correlated with outcome (P=0.0067). Thirty-nine percent of patients with shortened telomeres harbored telomere-related gene variants. Among 17 patients with shortened telomeres and high-resolution computed tomography features consistent with or probable UIP, 8 (47.1%) patients showed morphologic features compatible with UIP; multidisciplinary diagnosis most commonly was idiopathic pulmonary fibrosis (N=7, 41.2%) and familial pulmonary fibrosis (N=5, 29%) in these patients. In conclusion, patients with shortened telomeres have a spectrum of fILDs. They often demonstrate atypical and discordant features on pathology and radiology leading to diagnostic challenges.

Publication types

  • Comparative Study

MeSH terms

  • Adult
  • Aged
  • Biomarkers / analysis
  • Biopsy
  • Cyclin-Dependent Kinase Inhibitor p16 / analysis
  • Diagnosis, Differential
  • Female
  • Fibroblasts / chemistry
  • Fibroblasts / pathology
  • Humans
  • Immunohistochemistry
  • Lung Diseases, Interstitial* / diagnostic imaging
  • Lung Diseases, Interstitial* / genetics
  • Lung Diseases, Interstitial* / pathology
  • Lung* / chemistry
  • Lung* / diagnostic imaging
  • Lung* / pathology
  • Male
  • Middle Aged
  • Molecular Diagnostic Techniques*
  • Predictive Value of Tests
  • Retrospective Studies
  • Telomere / genetics
  • Telomere / pathology*
  • Telomere Shortening*
  • Tomography, X-Ray Computed*

Substances

  • Biomarkers
  • CDKN2A protein, human
  • Cyclin-Dependent Kinase Inhibitor p16