Immunomodulatory properties of characellide A on human peripheral blood mononuclear cells

Inflammopharmacology. 2021 Aug;29(4):1201-1210. doi: 10.1007/s10787-021-00836-5. Epub 2021 Jul 9.

Abstract

Marine sponges and their associated microbiota are multicellular animals known to produce metabolites with interesting pharmacological properties playing a pivotal role against a plethora of pathologic disorders such as inflammation, cancer and infections. Characellide A and B belong to a novel class of glycolipopeptides isolated from the deep sea marine sponge Characella pachastrelloides. In this study, we have evaluated the effects of characellide A and B on cytokine and chemokine release from human peripheral blood mononuclear cells (PBMC). Characellide A induces a concentration- and time-dependent CXCL8, IL-6 and TNF-α release from PBMC. This production is mediated by the induction of gene transcription. Moreover, cytokine/chemokine release induced by characellide A from PBMC is CD1d-dependent because a CD1d antagonist, 1,2-bis(diphenylphosphino)ethane [DPPE]-polyethylene glycolmonomethylether [PEG], specifically inhibits characellide A-induced activation of PBMC. In conclusion, characellide A is a novel modulator of adaptative/innate immune responses. Further studies are needed to understand its potential pharmacological application.

Keywords: CD1d; IL-6; LPS; Simplexide; TNF-α.

MeSH terms

  • Animals
  • Biological Factors / isolation & purification
  • Biological Factors / pharmacology*
  • Dose-Response Relationship, Drug
  • Humans
  • Immunomodulating Agents / isolation & purification
  • Immunomodulating Agents / pharmacology*
  • Immunomodulation / drug effects
  • Immunomodulation / physiology
  • Inflammation Mediators / agonists
  • Inflammation Mediators / immunology
  • Inflammation Mediators / metabolism*
  • Leukocytes, Mononuclear / drug effects*
  • Leukocytes, Mononuclear / immunology
  • Leukocytes, Mononuclear / metabolism*
  • Porifera*

Substances

  • Biological Factors
  • Immunomodulating Agents
  • Inflammation Mediators