Glucagon-(1-21) fails to inhibit meal size in rats

Peptides. 1987 Sep-Oct;8(5):943-5. doi: 10.1016/0196-9781(87)90085-4.

Abstract

Rats were intraperitoneally injected with 115 or 230 nmol/kg pancreatic glucagon or equimolar doses of glucagon-(1-21) just before refeeding at the beginning of the dark phase after a 12 hr period of food deprivation. Glucagon-(1-21) and pancreatic glucagon have been reported to have similar visceral effects, but glucagon-(1-21) does not have pancreatic glucagon's metabolic effects. In this experiment, both doses of pancreatic glucagon, but neither dose of glucagon-(1-21), significantly decreased meal size. This indicates that the C-terminal octapeptide of pancreatic glucagon is necessary for its satiety effect, just as it is for its metabolic effects.

Publication types

  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Animals
  • Dose-Response Relationship, Drug
  • Glucagon / pharmacology*
  • Male
  • Peptide Fragments / pharmacology*
  • Rats
  • Rats, Inbred Strains
  • Satiation / drug effects*
  • Structure-Activity Relationship

Substances

  • Peptide Fragments
  • glucagon (1-21)
  • Glucagon