Inherent genomic properties underlie the epigenomic heterogeneity of human induced pluripotent stem cells

Cell Rep. 2021 Nov 2;37(5):109909. doi: 10.1016/j.celrep.2021.109909.

Abstract

Human induced pluripotent stem cells (hiPSCs) show variable differentiation potential due to their epigenomic heterogeneity, whose extent/attributes remain unclear, except for well-studied elements/chromosomes such as imprints and the X chromosomes. Here, we show that seven hiPSC lines with variable germline potential exhibit substantial epigenomic heterogeneity, despite their uniform transcriptomes. Nearly a quarter of autosomal regions bear potentially differential chromatin modifications, with promoters/CpG islands for H3K27me3/H2AK119ub1 and evolutionarily young retrotransposons for H3K4me3. We identify 145 large autosomal blocks (≥100 kb) with differential H3K9me3 enrichment, many of which are lamina-associated domains (LADs) in somatic but not in embryonic stem cells. A majority of these epigenomic heterogeneities are independent of genetic variations. We identify an X chromosome state with chromosome-wide H3K9me3 that stably prevents X chromosome erosion. Importantly, the germline potential of female hiPSCs correlates with X chromosome inactivation. We propose that inherent genomic properties, including CpG density, transposons, and LADs, engender epigenomic heterogeneity in hiPSCs.

Keywords: DNA methylation; X chromosome inactivation; clonal heterogeneity; epigenome; histone modifications; human induced pluripotent stem cells; primordial germ cells.

Publication types

  • Comparative Study
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Cell Differentiation
  • Cell Line
  • Chromatin Assembly and Disassembly
  • Chromosomes, Human, X*
  • CpG Islands
  • DNA Transposable Elements
  • Epigenesis, Genetic*
  • Epigenome*
  • Epigenomics
  • Evolution, Molecular
  • Genetic Heterogeneity*
  • Histones / metabolism*
  • Humans
  • Induced Pluripotent Stem Cells / metabolism*
  • Methylation
  • Nuclear Lamina / genetics
  • Nuclear Lamina / metabolism
  • Protein Processing, Post-Translational
  • RNA, Long Noncoding / genetics
  • RNA, Long Noncoding / metabolism
  • X Chromosome Inactivation*

Substances

  • DNA Transposable Elements
  • Histones
  • RNA, Long Noncoding
  • XIST non-coding RNA