Phenotypic whole-cell screening identifies a protective carbohydrate epitope on Klebsiella pneumoniae

MAbs. 2022 Jan-Dec;14(1):2006123. doi: 10.1080/19420862.2021.2006123.

Abstract

The increasing global occurrence of recalcitrant multi-drug resistant Klebsiella pneumoniae infections warrants the investigation of alternative therapy options, such as the use of monoclonal antibodies (mAbs). We used a target-agnostic phage display approach to K. pneumoniae bacteria lacking bulky, highly variable surface polysaccharides in order to isolate antibodies targeting conserved epitopes among clinically relevant strains. One antibody population contained a high proportion of unique carbohydrate binders, and biolayer interferometry revealed these antibodies bound to lipopolysaccharide (LPS). Antibodies that bound to O1 and O1/O2 LPS were identified. Antibodies were found to promote opsonophagocytic killing by human monocyte-derived macrophages and clearance of macrophage-associated bacteria when assessed using high-content imaging. One antibody, B39, was found to protect mice in a lethal model of K. pneumoniae pneumonia against both O1 and O2 strains when dosed therapeutically. High-content imaging, western blotting and fluorescence-activated cell sorting were used to determine binding to a collection of clinical K. pneumoniae O1 and O2 strains. The data suggests B39 binds to D-galactan-I and D-galactan-II of the LPS of O1 and O2 strains. Thus, we have discovered an mAb with novel binding and functional activity properties that is a promising candidate for development as a novel biotherapeutic for the treatment and prevention of K. pneumoniae infections.

Keywords: Klebsiella pneumoniae; Phage display; antimicrobial resistance; lipopolysaccharide; monoclonal antibody.

Publication types

  • Research Support, Non-U.S. Gov't
  • Video-Audio Media

MeSH terms

  • Animals
  • Antibodies, Bacterial / immunology*
  • Drug Resistance, Multiple, Bacterial / genetics
  • Drug Resistance, Multiple, Bacterial / immunology
  • Epitopes / genetics
  • Epitopes / immunology*
  • Humans
  • Klebsiella Infections / genetics
  • Klebsiella Infections / immunology*
  • Klebsiella pneumoniae / genetics
  • Klebsiella pneumoniae / immunology*
  • Lipopolysaccharides / genetics
  • Lipopolysaccharides / immunology*
  • Macrophages / immunology*
  • Mice
  • Opsonization

Substances

  • Antibodies, Bacterial
  • Epitopes
  • Lipopolysaccharides

Grants and funding

This work was supported by the MedImmune; MedImmune.