BNIP3L/NIX-mediated mitophagy: molecular mechanisms and implications for human disease

Cell Death Dis. 2021 Dec 20;13(1):14. doi: 10.1038/s41419-021-04469-y.

Abstract

Mitophagy is a highly conserved cellular process that maintains the mitochondrial quantity by eliminating dysfunctional or superfluous mitochondria through autophagy machinery. The mitochondrial outer membrane protein BNIP3L/Nix serves as a mitophagy receptor by recognizing autophagosomes. BNIP3L is initially known to clear the mitochondria during the development of reticulocytes. Recent studies indicated it also engages in a variety of physiological and pathological processes. In this review, we provide an overview of how BNIP3L induces mitophagy and discuss the biological functions of BNIP3L and its regulation at the molecular level. We further discuss current evidence indicating the involvement of BNIP3L-mediated mitophagy in human disease, particularly in cancer and neurological disorders.

Publication types

  • Research Support, Non-U.S. Gov't
  • Review

MeSH terms

  • Amyotrophic Lateral Sclerosis / metabolism*
  • Animals
  • Autophagosomes / metabolism
  • Brain Injuries, Traumatic / metabolism*
  • Cerebral Hemorrhage / metabolism*
  • Humans
  • Membrane Proteins / metabolism*
  • Mitochondria / metabolism
  • Mitochondrial Membranes / metabolism
  • Mitochondrial Proteins / metabolism*
  • Mitophagy*
  • Neoplasms / metabolism*
  • Parkinson Disease / metabolism*
  • Proto-Oncogene Proteins / metabolism*
  • Signal Transduction*
  • Tumor Suppressor Proteins / metabolism*

Substances

  • BNIP3L protein, human
  • Membrane Proteins
  • Mitochondrial Proteins
  • Proto-Oncogene Proteins
  • Tumor Suppressor Proteins