Orchestrating serine/threonine phosphorylation and elucidating downstream effects by short linear motifs

Biochem J. 2022 Jan 14;479(1):1-22. doi: 10.1042/BCJ20200714.

Abstract

Cellular function is based on protein-protein interactions. A large proportion of these interactions involves the binding of short linear motifs (SLiMs) by folded globular domains. These interactions are regulated by post-translational modifications, such as phosphorylation, that create and break motif binding sites or tune the affinity of the interactions. In addition, motif-based interactions are involved in targeting serine/threonine kinases and phosphatases to their substrate and contribute to the specificity of the enzymatic actions regulating which sites are phosphorylated. Here, we review how SLiM-based interactions assist in determining the specificity of serine/threonine kinases and phosphatases, and how phosphorylation, in turn, affects motif-based interactions. We provide examples of SLiM-based interactions that are turned on/off, or are tuned by serine/threonine phosphorylation and exemplify how this affects SLiM-based protein complex formation.

Keywords: SLiM; kinase; modular domain; phosphatase; protein–protein interactions.

Publication types

  • Research Support, Non-U.S. Gov't
  • Review

MeSH terms

  • Binding Sites
  • Humans
  • Phosphoric Monoester Hydrolases / metabolism
  • Phosphorylation
  • Protein Interaction Domains and Motifs*
  • Protein Processing, Post-Translational
  • Protein Serine-Threonine Kinases / metabolism
  • Serine / chemistry*
  • Serine / metabolism*
  • Substrate Specificity
  • Threonine / chemistry*
  • Threonine / metabolism*

Substances

  • Threonine
  • Serine
  • Protein Serine-Threonine Kinases
  • Phosphoric Monoester Hydrolases