Dynamic interplay between the periplasmic chaperone SurA and the BAM complex in outer membrane protein folding

Commun Biol. 2022 Jun 8;5(1):560. doi: 10.1038/s42003-022-03502-w.

Abstract

Correct folding of outer membrane proteins (OMPs) into the outer membrane of Gram-negative bacteria depends on delivery of unfolded OMPs to the β-barrel assembly machinery (BAM). How unfolded substrates are presented to BAM remains elusive, but the major OMP chaperone SurA is proposed to play a key role. Here, we have used hydrogen deuterium exchange mass spectrometry (HDX-MS), crosslinking, in vitro folding and binding assays and computational modelling to show that the core domain of SurA and one of its two PPIase domains are key to the SurA-BAM interaction and are required for maximal catalysis of OMP folding. We reveal that binding causes changes in BAM and SurA conformation and/or dynamics distal to the sites of binding, including at the BamA β1-β16 seam. We propose a model for OMP biogenesis in which SurA plays a crucial role in OMP delivery and primes BAM to accept substrates for folding.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Bacterial Outer Membrane Proteins / metabolism
  • Carrier Proteins / metabolism
  • Escherichia coli / metabolism
  • Escherichia coli Proteins* / metabolism
  • Molecular Chaperones / metabolism
  • Peptidylprolyl Isomerase / metabolism
  • Periplasm / metabolism
  • Protein Folding

Substances

  • Bacterial Outer Membrane Proteins
  • BamA protein, E coli
  • Carrier Proteins
  • Escherichia coli Proteins
  • Molecular Chaperones
  • SurA protein, E coli
  • Peptidylprolyl Isomerase