miR-199a Is Upregulated in GDM Targeting the MeCP2-Trpc3 Pathway

Front Endocrinol (Lausanne). 2022 Jul 14:13:917386. doi: 10.3389/fendo.2022.917386. eCollection 2022.

Abstract

Gestational diabetes mellitus (GDM), the most common medical pregnancy complication, has become a growing problem. More and more studies have shown that microRNAs are closely related to metabolic processes. The purpose of this paper is to investigate the role of up-regulation of miR-199a-5p expression in GDM. We found that miR-199a-5p was significantly up-regulated in the placenta of GDM patients compared with normal pregnant women, and expressed in placental villi. miR-199a-5p can regulate the glucose pathway by inhibiting the expression of methyl CpG-binding protein 2 (MeCP2) and down-regulating canonical transient receptor potential 3 (Trpc3). This suggests that miR-199a-5p may regulate the glucose pathway by regulating methylation levels, leading to the occurrence of GDM.

Keywords: MeCP2; MiR-199a; TRPC3; gestational diabetes mellitus; placenta.

MeSH terms

  • Diabetes, Gestational* / genetics
  • Diabetes, Gestational* / metabolism
  • Female
  • Glucose / metabolism
  • Humans
  • MicroRNAs* / genetics
  • MicroRNAs* / metabolism
  • Placenta / metabolism
  • Pregnancy
  • Up-Regulation

Substances

  • MicroRNAs
  • mirn199 microRNA, human
  • Glucose